Embolism of Central Retinal Artery

Quantitative analysis of retinal layers' optical intensities on 3D optical coherence tomography for central retinal artery occlusion

“…The ONL, photoreceptor, RPE and choroidal layers are not supplied by the retinal artery and its branches. Animal and human autopsy studies previously showed that the outer retina does not change in CRAO 24 26 . Our results found that the optical intensities in the outer retina from the photoreceptor layer to the choroid were reduced in the acute CRAO phase, especially in the photoreceptor and RPE layers.…”

“…The blood supply of RNFL is also provided from the central retinal artery system. In a pathological study of human autopsy eyes with CRAO, severe edema of the RNFL was frequently noted 26 . It is conceivable that edema of RNFL does not affect its OCT optical intensity.…”

Quantitative analysis of retinal layers' optical intensities on 3D optical coherence tomography for central retinal artery occlusion

“…The ONL, photoreceptor, RPE and choroidal layers are not supplied by the retinal artery and its branches. Animal and human autopsy studies previously showed that the outer retina does not change in CRAO 24 26 . Our results found that the optical intensities in the outer retina from the photoreceptor layer to the choroid were reduced in the acute CRAO phase, especially in the photoreceptor and RPE layers.…”

“…The blood supply of RNFL is also provided from the central retinal artery system. In a pathological study of human autopsy eyes with CRAO, severe edema of the RNFL was frequently noted 26 . It is conceivable that edema of RNFL does not affect its OCT optical intensity.…”

Progress in central retinal artery occlusion: a narrative review

“… 19 , 20 Platelet-fibrin emboli are mobile, dull, grayish-white entities that originate from carotid and intracardiac thromboses. 21 , 22 Calcified emboli are white solid entities with an oval shape that typically originate from calcified aortic stenosis; they also occur in patients with mitral valve disease or aortic valve disease. 23 , 24 Periarteriolar sheathing, a post-embolic phenomenon in some patients with cholesterol or calcified CRAO, is regarded as a giant cell reaction and a type of injury-induced endothelial cell proliferation.…”

The Association between Elevated Hematocrit and Retinal Artery Occlusion in Adult Patients

“…According to previous literature, both thrombi and emboli could cause myocardial infarction (MI) [15,16] and ischemic stroke [17,18]. The relationship between RAO and stroke [4,19] or MI [4,20,21] has been suggested. Thus, we may use the association of Hct and MI as well as ischemic stroke to assume the correlation between Hct and RAO, the severe retinal embolic disease.…”

Quantitative analysis of retinal layers' optical intensities on 3D optical coherence tomography for central retinal artery occlusion

“…The ONL, photoreceptor, RPE and choroidal layers are not supplied by the retinal artery and its branches. Animal and human autopsy studies previously showed that the outer retina does not change in CRAO 24 26 . Our results found that the optical intensities in the outer retina from the photoreceptor layer to the choroid were reduced in the acute CRAO phase, especially in the photoreceptor and RPE layers.…”

“…The blood supply of RNFL is also provided from the central retinal artery system. In a pathological study of human autopsy eyes with CRAO, severe edema of the RNFL was frequently noted 26 . It is conceivable that edema of RNFL does not affect its OCT optical intensity.…”

Progress in central retinal artery occlusion: a narrative review

“… 19 , 20 Platelet-fibrin emboli are mobile, dull, grayish-white entities that originate from carotid and intracardiac thromboses. 21 , 22 Calcified emboli are white solid entities with an oval shape that typically originate from calcified aortic stenosis; they also occur in patients with mitral valve disease or aortic valve disease. 23 , 24 Periarteriolar sheathing, a post-embolic phenomenon in some patients with cholesterol or calcified CRAO, is regarded as a giant cell reaction and a type of injury-induced endothelial cell proliferation.…”

The Association between Elevated Hematocrit and Retinal Artery Occlusion in Adult Patients

“…According to previous literature, both thrombi and emboli could cause myocardial infarction (MI) [15,16] and ischemic stroke [17,18]. The relationship between RAO and stroke [4,19] or MI [4,20,21] has been suggested. Thus, we may use the association of Hct and MI as well as ischemic stroke to assume the correlation between Hct and RAO, the severe retinal embolic disease.…”

Quantitative analysis of retinal layers' optical intensities on 3D optical coherence tomography for central retinal artery occlusion

“…The ONL, photoreceptor, RPE and choroidal layers are not supplied by the retinal artery and its branches. Animal and human autopsy studies previously showed that the outer retina does not change in CRAO 24 26 . Our results found that the optical intensities in the outer retina from the photoreceptor layer to the choroid were reduced in the acute CRAO phase, especially in the photoreceptor and RPE layers.…”

“…The blood supply of RNFL is also provided from the central retinal artery system. In a pathological study of human autopsy eyes with CRAO, severe edema of the RNFL was frequently noted 26 . It is conceivable that edema of RNFL does not affect its OCT optical intensity.…”

Progress in central retinal artery occlusion: a narrative review

“… 19 , 20 Platelet-fibrin emboli are mobile, dull, grayish-white entities that originate from carotid and intracardiac thromboses. 21 , 22 Calcified emboli are white solid entities with an oval shape that typically originate from calcified aortic stenosis; they also occur in patients with mitral valve disease or aortic valve disease. 23 , 24 Periarteriolar sheathing, a post-embolic phenomenon in some patients with cholesterol or calcified CRAO, is regarded as a giant cell reaction and a type of injury-induced endothelial cell proliferation.…”

The Association between Elevated Hematocrit and Retinal Artery Occlusion in Adult Patients

“…According to previous literature, both thrombi and emboli could cause myocardial infarction (MI) [15,16] and ischemic stroke [17,18]. The relationship between RAO and stroke [4,19] or MI [4,20,21] has been suggested. Thus, we may use the association of Hct and MI as well as ischemic stroke to assume the correlation between Hct and RAO, the severe retinal embolic disease.…”