Elevated values of tri-iodothyronine in victims of sudden infant death syndrome

Chacon, Marco; Tildón, J. Tyson

doi:10.1016/s0022-3476(81)80406-4

Cited by 44 publications

(1 citation statement)

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“…The accelerated maturation hypothesis proposes that early maturation for sleep and waking results in a higher threshold for arousal at a critical time. It is supported by the association of an increased thyroid hormone concentration, a marker of accelerated neurological development that has been reported in infants dying from SIDS.32 33 The delayed maturation hypothesis is also supported histologically by the observation of the persistence of reticular spine density in the brainstem of SIDS babies which normally decreases 34 weeks after conception.6 This hypothesis is also supported by the findings of this study.…”

Section: Discussionsupporting

confidence: 68%

Fetal behaviour and the sudden infant death syndrome (SIDS).

Smoleniec

James

1995

Archives of Disease in Childhood - Fetal and Neonatal Edition

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To examine whether differences in sleep maturation could be identified before birth, behavioural studies were carried out in 28 fetuses. Studies were possible in all 28 fetuses at 28 weeks, but only in 26 fetuses at 36 weeks (two fetuses delivered before 36 weeks). The risk ofsudden infant death syndrome (SIDS) was determined using the Oxford SIDS scoring system. The fetuses at greater risk of SIDS had coincidence ofbehavioural characteristics for a significantly lower percentage of the time than those at low risk. This difference reached significance (p-005) only at 36 weeks. (Arch Dis Child 1995; 72: F168-F171)

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Section: Discussionsupporting

confidence: 68%