Elevated Amount of Human Papillomavirus 31 DNA in a Squamous Cell Carcinoma Developed from Bowenoid Papulosis

Hama, Naoto; Ohtsuka, Tsutomu; Yamazaki, Soji

doi:10.1159/000080858

Cited by 13 publications

(11 citation statements)

References 26 publications

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“…HPV types 31, 32, 34, 39, 42, 48, and 51-54 have also been found in BP lesions at a greater frequency than in normal controls. [66][67][68][69][70][71][72][73][74][75][76][77] In addition, the presence of viral particles in BP tumor cells supports the role of HPV as an etiologic agent, although viral inclusions are not present in all cases. [77][78][79][80] These findings suggest a mechanism of viral pathogenesis.…”

Section: Bowenoid Papulosismentioning

confidence: 99%

Viral-Associated Nonmelanoma Skin Cancers: A Review

Dubina¹,

Goldenberg²

2009

The American Journal of Dermatopathology

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Nonmelanoma skin cancers are the most common malignancies in the United States, with over 1.3 million patients diagnosed yearly. Several types of nonmelanoma skin cancer and precancerous lesions have an associated viral pathogenesis, including epidermodysplasia verruciformis, verrucous carcinoma, bowenoid papulosis, Kaposi sarcoma, squamous cell carcinoma, and, most recently, Merkel cell carcinoma. It is now widely accepted that high-risk human papillomaviruses (HPVs) play a key role in pathogenesis of cervical and anogenital cancer. Currently, 108 types of HPV have been discovered. They can be divided into genera including alpha, beta, and gamma HPVs, which comprise the majority of cutaneous HPVs. The relationship between viruses and cancer is a popular focus of research in an era of AIDS and organ transplantation, where immunosuppression is not uncommon. The incidence of viral-related malignancies in these populations is increasing. Because of the widespread nature of these cutaneous malignancies, an evaluation of these conditions is valuable. This review of the literature will focus on the histologic aspects of viral-associated skin malignancies, as well as the epidemiology, etiology, and clinical aspects of these diseases.

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Section: Bowenoid Papulosismentioning

confidence: 99%

Viral-Associated Nonmelanoma Skin Cancers: A Review

Dubina¹,

Goldenberg²

2009

The American Journal of Dermatopathology

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“…HPV type 16 and 18 may represent a high risk for cervical and vulvar neoplasia [35]. For this reason, typing of the HPV and clinical follow-up are desirable [36]. Swabbing with acetic acid 5% facilitates the determination of the extent of the lesions.…”

Section: Precancerous Lesionsmentioning

confidence: 99%

Dermatoses of the Male Genital Area

Eichmann¹

2005

Dermatology

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This is an overview of dermatoses which are predominantly seen in the genital area. Five large groups of dermatoses can be distinguished: inflammatory dermatoses without any causative organism, infections and dermatoses originating from cutaneous appendages, precancerous lesions, pigmented lesions and genital pruritus. The etiology, causative factors, clinical features and newer treatment options are considered. Because of local environmental factors, genital dermatoses often present in a different way than in other localizations.

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“…Hama et al. reported a case in which SCC arose from Bowenoid papulosis 7 . The SCC was poorly differentiated and exhibited the features of Bowen’s carcinoma 7 .…”

Section: Introductionmentioning

confidence: 99%