volume 66, issue 6, P3306-3315 1992
DOI: 10.1128/jvi.66.6.3306-3315.1992
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Abstract: The role of the cytoplasmic domain of the human immunodeficiency virus type 1 (HIV-1) envelope glycoproteins in virus replication was investigated. Deletion of residues 840 to 856 at the carboxyl terminus of gp4l reduced the efficiency of virus entry during an early step in the virus life cycle between CD4 binding and formation of the DNA provirus without affecting envelope glycoprotein synthesis, processing, or syncytiumforming ability. Deletion of residues amino terminal to residue 846 was associated with de…

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