Archives of Otolaryngology - Head and Neck Surgery
 1953
DOI: 10.1001/archotol.1953.00710040079008
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Effect of Prolonged Exposure to Intense Noise on Hearing Acuity

J. SATALOFF1
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Cited by 14 publications

(3 citation statements)
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“…(Talbott et al, 1990), among others. Our results also concur with those of studies that posit that the harmful effect of noise is proportional to the duration of the exposure, such as Clemente Ibáñez (Clemente Ibáñez, 1991), Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995).…”
Section: Discussion
supporting
confidence: 92%
“…Other studies also agree and predict that occupational hypoacusia will more often be present in old individuals than in young ones (Delgado, 1991;Mcshane, Hyde, Finkelstein, & Alberti, 1991). There are likewise studies that propose that the harmful effect of noise is proportional to the duration of the exposure (Clemente Ibáñez, 1991), with many authors agreeing with this conclusion, including Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995). We can state that there is a direct relationship between the length of the noise exposure and the presence of hearing loss, especially at 4,000 Hz and nearby frequencies (Burns & Robinson, 1970;Dobie, 1995;Howell, 1978;Sataloff, 1953).…”
Section: Framework Relationship
mentioning
confidence: 83%
“…There are likewise studies that propose that the harmful effect of noise is proportional to the duration of the exposure (Clemente Ibáñez, 1991), with many authors agreeing with this conclusion, including Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995). We can state that there is a direct relationship between the length of the noise exposure and the presence of hearing loss, especially at 4,000 Hz and nearby frequencies (Burns & Robinson, 1970;Dobie, 1995;Howell, 1978;Sataloff, 1953). As for the form in which deafness or hypoacusia occurs, there is evidence that over the long term, noiseinduced hearing damage is caused by the gradual accumulation of microtraumas due to noise that, little by little, result in the degradation of hearing ability (Gravendeel & Plomp, 1960).…”
Section: Framework Relationship
mentioning
confidence: 95%
See 2 more Smart Citations
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…(Talbott et al, 1990), among others. Our results also concur with those of studies that posit that the harmful effect of noise is proportional to the duration of the exposure, such as Clemente Ibáñez (Clemente Ibáñez, 1991), Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995).…”
Section: Discussion
supporting
confidence: 92%
“…Other studies also agree and predict that occupational hypoacusia will more often be present in old individuals than in young ones (Delgado, 1991;Mcshane, Hyde, Finkelstein, & Alberti, 1991). There are likewise studies that propose that the harmful effect of noise is proportional to the duration of the exposure (Clemente Ibáñez, 1991), with many authors agreeing with this conclusion, including Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995). We can state that there is a direct relationship between the length of the noise exposure and the presence of hearing loss, especially at 4,000 Hz and nearby frequencies (Burns & Robinson, 1970;Dobie, 1995;Howell, 1978;Sataloff, 1953).…”
Section: Framework Relationship
mentioning
confidence: 83%
“…There are likewise studies that propose that the harmful effect of noise is proportional to the duration of the exposure (Clemente Ibáñez, 1991), with many authors agreeing with this conclusion, including Sataloff (Sataloff, 1953), Howell (Howell, 1978), Burns and Robinson (Burns & Robinson, 1970) and Dobie (Dobie, 1995). We can state that there is a direct relationship between the length of the noise exposure and the presence of hearing loss, especially at 4,000 Hz and nearby frequencies (Burns & Robinson, 1970;Dobie, 1995;Howell, 1978;Sataloff, 1953). As for the form in which deafness or hypoacusia occurs, there is evidence that over the long term, noiseinduced hearing damage is caused by the gradual accumulation of microtraumas due to noise that, little by little, result in the degradation of hearing ability (Gravendeel & Plomp, 1960).…”
Section: Framework Relationship
mentioning
confidence: 95%
See 1 more Smart Citation
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
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