A 73 year old man developed a left ventricular pseudoaneurysm following acute myocardial infarction. Coronary angiography showed triple vessel disease with total occlusion of the right coronary artery. On left ventriculography, a serpentine-like pseudoaneurysm was demonstrated that originated from the posterobasal wall of the left ventricle and extended to the right ventricular free wall. He underwent coronary artery bypass surgery with no plication of the pseudoaneurysm. An organised thrombus was also found within the cavity of the pseudoaneurysm. He was doing well approximately eight months after the operation. The prognosis might be determined by the organised thrombus, the serpentinelike structure of pseudoaneurysm, the coronary revascularisation, and the vigorous medical management. (Heart 1998;80:94-97) Keywords: acute myocardial infarction; pseudoaneurysm; coronary artery bypass surgery Left ventricular pseudoaneurysm is a rare complication of acute myocardial infarction. It occurs as a consequence of rupture of the ventricular free wall and is confined by a portion of pericardium. Early surgical intervention is recommended because there is a risk of rupture even with a small pseudoaneurysm. 1 An electrical or mechanical event is usually the final cause of death. We report a patient who survived with pseudoaneurysm eight months following acute myocardial infarction.
Case reportA 73 year old man was admitted to hospital on 11 January 1997 because of prolonged chest pain of 14 hours. Physical examination revealed a heart rate of 97 beats/min, a respiratory rate of 16 breaths/min, and a blood pressure of 154/106 mm Hg. He had no history of cardiovascular disease. No carotid bruit was heard. Auscultation of the lungs showed rales over the right basal area. There was no murmur, gallop, or friction rub. Other physical findings were unremarkable. Chest radiography was essentially normal. Acute inferior myocardial infarction was diagnosed by typical evolutional changes in a 12 lead electrocardiogram and raised creatine kinase (up to 3746 IU/l with MB form of 8.5%). There was no evidence of right ventricular infarction by initial right sided electrocardiogram. Hypotension (blood pressure 85/45 mm Hg) developed two days later and he was managed with aspirin, isosorbide dinitrate, and intravenous dopamine. Transthoracic cross sectional echocardiography (Sonos 2500; Hewlett-Packard, Andover, Massachusetts, USA) was done immediately to detect the possible aetiologies of hypotension. It showed inferior and posterior left ventricular wall hypokinesis and right ventricular free wall akinesis with mild mitral regurgitation. There was no pericardial eVusion, intracardiac shunt, or cardiac rupture.The patient underwent cardiac catheterisation, coronary angiography, and left ventriculography 12 days after admission. Coronary angiography showed triple vessel disease with total occlusion of the right coronary artery. Left ventriculography showed diaphragmatic, posterobasal, apical septal, and posterolateral akinesis...