2001
DOI: 10.1016/s0378-1097(01)00450-5 View full text |Buy / Rent full text
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Abstract: TNF/LTK deficient mice are devoid of Peyer's patches and lack mesenteric lymph nodes. Translocation, especially in the early steps after intragastric delivery of Listeria monocytogenes, has been explored in this study, and the role of TNFK has been addressed. We showed that L. monocytogenes translocation occurred at least as efficiently in TNF/LTK3/3 mice as in TNF/LTK+/+ littermates. Even very low inocula (2.7U10 4 cfu) could initiate infection in the TNF/LTK deficient mice. Early kinetics of dissemination to… Show more

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“…MC release a wide variety of cytokines and chemokines in response to L.m, including TNF-a, IL-1b, IL-6, IL-13 and MCP-1 (15,16,33). Interestingly, TNF-a or TNF-receptor deficient mice are highly susceptible to L.m infection (51)(52)(53). This could be associated with the TNF-a exacerbation of intracellular killing of L.m by macrophages through ROS and nitric oxide production (54,55).…”
Section: Discussionmentioning
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“…MC release a wide variety of cytokines and chemokines in response to L.m, including TNF-a, IL-1b, IL-6, IL-13 and MCP-1 (15,16,33). Interestingly, TNF-a or TNF-receptor deficient mice are highly susceptible to L.m infection (51)(52)(53). This could be associated with the TNF-a exacerbation of intracellular killing of L.m by macrophages through ROS and nitric oxide production (54,55).…”
Section: Discussionmentioning