Detection of Missing Vesicoureteral Reflux with Bethanechol Chloride-Aided Voiding Cystourethrography

Koyanagi, Tomohiko; Morita, Hajime; Gotoh, Tamihiro; Nonomura, Katsuya; Terabe, Masaki; Nantani, Masami

doi:10.1016/s0022-5347(17)40900-1

Cited by 4 publications

(2 citation statements)

References 13 publications

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“…8,37,38 Morita et al reported in diabetic patients that there was a greater chance of detecting denervation supersensitivity of the urethra to an a-sympathetic agonist than of the bladder. 33,39 This implies that the isolated neurogenic dysfunction of the urethra, as evidenced by denervation supersensitivity in the absence of vesical denervation supersensitivity (Vds), can cause voiding dysfunction in diabetic patients. 33,39 A correlation between diabetic cystopathy and abnormal sympathetic skin response has been reported.…”

Section: Diabetic Neuropathymentioning

confidence: 99%

“…33,39 This implies that the isolated neurogenic dysfunction of the urethra, as evidenced by denervation supersensitivity in the absence of vesical denervation supersensitivity (Vds), can cause voiding dysfunction in diabetic patients. 33,39 A correlation between diabetic cystopathy and abnormal sympathetic skin response has been reported. 23 Sympathetic skin responses are mediated by unmyelinated postganglionic sympathetic fibers.…”

Section: Diabetic Neuropathymentioning

confidence: 99%

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Bladder dysfunction in peripheral neuropathies

et al. 2011

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Normal bladder function depends on the complex interaction of sensory and motor pathways. Bladder dysfunction can develop as a result of several neurological conditions. It can happen in a number of ways, including diabetic cystopathy, detrusor overactivity, bladder outlet obstruction, and urge and stress urinary incontinence. Diabetic neuropathy is the most common cause of peripheral neuropathy-associated bladder dysfunction. Guillain-Barré syndrome (GBS), human immunodeficiency virus (HIV)-associated neuropathy, chronic inflammatory demyelinating polyneuropathy (CIDP), and amyloid neuropathy are other major causes. The diagnosis of bladder dysfunction should be established by the history of neurological symptoms, neurological examination, and urological evaluation. Functional evaluation of the lower urinary tract includes cystometry, sphincter electromyography, uroflowmetry, and urethral pressure profilometry. Management of urinary symptoms in patients with bladder dysfunction is usually supportive. In some cases, alpha-blocker and/or anti-muscarinic agents are needed to help improve urinary dysfunction. Intermittent self-catheterization is needed occasionally for patients with slow and/or poor recovery.

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