2002
DOI: 10.1783/147118902101195910
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Depo-Provera<SUP>®</SUP> and bone density

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Cited by 21 publications
(1 citation statement)
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References 41 publications
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“…including secondary causes of osteoporosis) were collected from the electronic medical record. Medications that decrease bone mineral density include: glucocorticoids (oral and inhaled), antiasthma (only budesonide/formoterol, fluticasone/salmeterol), anti-inflammatory (inhaled, including beclomethasone dipropionate, budesonide, ciclesonide, flunisolide, fluticasone, mometasone), anticonvulsants (only divalproex, phenytoin, carbamazepine), chemotherapeutic agents, anticoagulants, and depo-provera (Van Staa et al 2003; Chee et al 2014; Drummond et al 2008; Lee et al 2010; Brufsky et al 2008; Rey-Sanchez et al 2011; Gbolade 2002). Medications that increase bone mineral density include: denosumab, progestin only, contraceptive (systemic, levonorgesterel), bisphosphonates, androgen/anabolics (testosterone, topical or transdermal), and estrogen (transdermal, oral) (Cummings et al 2009; Caird et al 1994; Munk-Jensen et al 1988; Russell et al 2011; Rodriguez-Tolra et al 2013; Manolagas et al 2002).…”
Section: Methodsmentioning
confidence: 99%
“…including secondary causes of osteoporosis) were collected from the electronic medical record. Medications that decrease bone mineral density include: glucocorticoids (oral and inhaled), antiasthma (only budesonide/formoterol, fluticasone/salmeterol), anti-inflammatory (inhaled, including beclomethasone dipropionate, budesonide, ciclesonide, flunisolide, fluticasone, mometasone), anticonvulsants (only divalproex, phenytoin, carbamazepine), chemotherapeutic agents, anticoagulants, and depo-provera (Van Staa et al 2003; Chee et al 2014; Drummond et al 2008; Lee et al 2010; Brufsky et al 2008; Rey-Sanchez et al 2011; Gbolade 2002). Medications that increase bone mineral density include: denosumab, progestin only, contraceptive (systemic, levonorgesterel), bisphosphonates, androgen/anabolics (testosterone, topical or transdermal), and estrogen (transdermal, oral) (Cummings et al 2009; Caird et al 1994; Munk-Jensen et al 1988; Russell et al 2011; Rodriguez-Tolra et al 2013; Manolagas et al 2002).…”
Section: Methodsmentioning
confidence: 99%