Dental erosion, gastro-oesophageal reflux disease and saliva: how are they related?

Dental erosion, an extraesophageal manifestation of gastroesophageal reflux disease: the experience of a center for digestive physiology in Southeastern Mexico

“…It has been reported that the prevalence of dental erosion in patients with GERD varies from 5.00 % to 58.41 % (8,24-34), which we corroborated in our series of patients, were a prevalence of 78.9 % was found, higher than the prevalence reported by other authors (8,28,30,31). This is directly related to the severity and duration of the disease; in our cohort, the average time of evolution was 50.09 ± 32.26 months, and the presence of acid reflux was demonstrated in 70.0 % of cases by ph-metry (8,(19)(20)(21)(22)24,34). By comparing the degree of dental erosion with the severity of esophagitis, we found that 75.0 % of the patients with grade N0 and N1 had normal mucosa or grade A esophagitis, whereas patients with N2 and N3 DE were associated with greater frequency to esophagitis stages C and D; the difference was statistically significant (p = 0.021).…”

“…The location of this damage in patients with GERD is more common in the occlusal and palatal surfaces of the upper teeth and in the buccal and lingual/occlusal surfaces of the lower teeth, because the position of the tongue leads the acid to these surfaces (8)(9)(10).…”

“…Different population groups have reported an association of GERD with dental erosion, the prevalence of which ranges from 5 % to 58.41 %, as shown in table I (8,(26)(27)(28)(29)(30)(31)(32)(33). In 2012, Vargas et al published a series of 150 cases of Latin American patients and the two conditions were associated in 30 % (34) of them.…”

Oral Cavity and Systemic Diseases—Gastroesophageal Reflux Disease

“…The most frequent oral manifestation of GERD is erosion of tooth enamel (2,3,19,23). The pH of the gastric acid is much lower than the critical pH of enamel dissolution, therefore, reflux of stomach contents into the oral cavity over an extended period of time can cause severe loss of tooth structure (15,16).…”

Effects of simulated gastric juice on color stability, surface roughness and microhardness of laboratory-processed composites

“…The gastric contents affect the oral cavity and teeth as a result of gastroesophageal reflux, since pH of the gastric acid ranges between 1 and 1.5 -far below the critical pH of 5.5 at which tooth enamel will dissolve 2) . Gastric juice has been shown to demineralize enamel, dentin, and root cementum in in vivo and in vitro studies [3][4][5][6] . Laboratory-processed indirect resin composite systems may be the solution to some of the problems inherent to dental ceramics.…”