2011
DOI: 10.1016/j.cmet.2011.09.005
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Abstract: In the above article, we unfortunately miscited the findings of Toivonen et al. (2007) in our Discussion, where we wrote: ''In addition, caloric restriction does not increase life span further in flies with reduced Indy expression (Toivonen et al., 2007; Wang et al., 2009), pointing to similar underlying mechanisms in both conditions.'' Toivonen et al. did not investigate caloric restriction and did not see an effect for Indy on Drosophila life span. However, further studies by Wang et al. (2009) supported a r… Show more

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Cited by 53 publications
(102 citation statements)
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“…This is supported by metabolic studies in Slc13a5-null mice, which show that the absence the transporter protects against dietinduced adiposity, insulin resistance, and the metabolic syndrome (Birkenfeld et al, 2011).…”
Section: Discussionsupporting
confidence: 49%
See 1 more Smart Citation
“…This is supported by metabolic studies in Slc13a5-null mice, which show that the absence the transporter protects against dietinduced adiposity, insulin resistance, and the metabolic syndrome (Birkenfeld et al, 2011).…”
Section: Discussionsupporting
confidence: 49%
“…Citrate entering into the cytoplasm of the hepatocytes via NaCT is avidly used for the synthesis of fatty acids (Inoue et al, 2003;Ganapathy and Fei, 2005). As such, deletion of the transporter in mice protects against diet-induced insulin resistance and metabolic syndrome (Birkenfeld et al, 2011). In the testes, NaCT is expressed in sperm cells (P. M. Martin and V. Ganapathy, unpublished data).…”
Section: Introductionmentioning
confidence: 99%
“…Loss of SLC13A5 augments energy expenditure and hepatic fat oxidation and attenuates hepatic lipogenesis, reducing overall growth (18). SLC13A5 was hypermethylated and downregulated in SGA placenta and cord blood.…”
Section: Discussionmentioning
confidence: 95%
“…Essential to this process are the AMPK-mediated activation of PGC-1a and SIRT1. AMPK can directly phosphorylate and activate PGC-1a (Jager et al 2007); other pathways that activate AMPK can result in a subsequent increase in PGC-1a protein expression and mitochondrial biogenesis (Birkenfeld et al 2011). AMPK through Nampt-dependent and -independent mechanisms is also able to increase NAD þ levels, which promotes SIRT1 activity and the deacetylation/activation of PGC1a ).…”
Section: Amp-activated Protein Kinase (Ampk)mentioning
confidence: 99%