Cutaneous periarteritis nodosa. Hepatitis B surface antigen-containing immunocomplexes and polymorphonuclear-leukocyte lysosomal enzyme release

Pette, J

doi:10.1001/archderm.120.1.109

Cited by 9 publications

(7 citation statements)

References 9 publications

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“…Cases of CPN associated with inflammatory bowel disease or hepatitis antigenemia have been described recently (36)(37)(38)(39)(40)(41)(42) and immune complexes have been suggested as having an important role in the pathogenesis of the disease (39)(40)(41)(42). In this study, the serologic studies for viral hepatitis and circulating immune complexes of the examined patients were negative or normal.…”

Section: Immunohistochemical Studiesmentioning

confidence: 57%

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Cutaneous Polyarteritis Nodosa: A Clinical and Histopathological Study of 20 Cases

Chen

1989

The Journal of Dermatology

122

102

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Twenty cases diagnosed as cutaneous polyarteritis nodosa (CPN) and confirmed by skin biopsy over the last 17 years were reviewed in our department. Based upon their clinical features, laboratory findings, and long-term observation of the disease course, they were divided into three groups. 1) Group 1 comprised 16 cases which were classified as the mild cutaneous form. The disease was confined to the skin with occasional involvement of peripheral nerves and skeletal muscles of the affected extremity. They generally followed a benign course. 2) Group 2 comprised 2 cases classified as the severe form. Despite severe clinical manifestations and several abnormal laboratory findings, the disease was limited to the skin, muscles, and peripheral nerves without any visceral involvement over follow-up periods of 11 years and 5 years, respectively. 3) Group 3 comprised 2 cases of the progressive form; in these the disease had begun with a cutaneous lesion and progressed to the systemic form after 19 and 18 year periods of recurrent episodes of cutaneous lesions, respectively. One died of gastrointestinal bleeding. In group 3, serum antinuclear antibodies and rheumatoid factor were positive. The autoimmune mechanism seems to play a role in this group. It is clear from the results of this study that not all patients whose vasculitic lesions are apparently limited to the skin remain in a benign course. Long-term follow-up is essential.

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Section: Immunohistochemical Studiesmentioning

confidence: 57%

“…Besides hepatitis B virus (41,42), other infectious agents such as Streptococcus (6,43) and Mycobacterium tuberculosis (29) have also been implicated in the pathogenesis of CPN. None of these agents were found in this study.…”

Section: Immunohistochemical Studiesmentioning

confidence: 99%

Cutaneous Polyarteritis Nodosa: A Clinical and Histopathological Study of 20 Cases

Chen

1989

The Journal of Dermatology

122

102

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“…Until today, the etiology of CPN has remained unknown. Some authors suggest an association to hepatitis B, 12,13 virus infections or other infectious agents such as streptococcus 8,14 or mycobacterium tuberculosis 15 . Associations with other inflammatory disorders like polychondritis 16 and ulcerative colitis and Crohn's disease 17,18 have been reported.…”

Section: Discussionmentioning

confidence: 99%

Panarteritis cutanea benigna – an entity limited to the skin or cutaneous presentation of a systemic necrotizing vasculitis? Report of seven cases and review of the literature

Rogalski¹,

Sticherling²

2007

Int J Dermatology

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The etiology of panarteritis nodosa cutanea is unknown in detail. Both focal infections and hypersensitivity reactions are discussed. Differential diagnosis includes systemic panarteritis, livedo reticularis, Sneddon's syndrome or erythema nodosum. Despite the prognostically favorable but prolonged chronic course of the disease over decades, patients should be regularly examined to exclude possible transition to systemic disease.

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“…Unlike systemic PAN, where up to 30–50% of cases are associated with chronic active hepatitis B infection, 1,2 cutaneous PAN is distinct in that it is usually not associated with hepatitis B infection 3,4 . Furthermore, while there have been reports of cutaneous PAN associated with the wild‐type virus, 5,6 our case was instead associated with the precore mutant virus.…”

mentioning

confidence: 65%

Cutaneous polyarteritis nodosa associated with precore mutant hepatitis B infection

Tang¹,

Liew²,

Ng³

et al. 2003

Br J Dermatol

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Cutaneous periarteritis nodosa. Hepatitis B surface antigen-containing immunocomplexes and polymorphonuclear-leukocyte lysosomal enzyme release

Cited by 9 publications

References 9 publications

Cutaneous Polyarteritis Nodosa: A Clinical and Histopathological Study of 20 Cases

Cutaneous Polyarteritis Nodosa: A Clinical and Histopathological Study of 20 Cases

Panarteritis cutanea benigna – an entity limited to the skin or cutaneous presentation of a systemic necrotizing vasculitis? Report of seven cases and review of the literature

Cutaneous polyarteritis nodosa associated with precore mutant hepatitis B infection

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