2006
DOI: 10.1152/physrev.00029.2005
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Abstract: The cardiac muscle and the coronary vasculature are in close proximity to each other, and a two-way interaction, called cross-talk, exists. Here we focus on the mechanical aspects of cross-talk including the role of the extracellular matrix. Cardiac muscle affects the coronary vasculature. In diastole, the effect of the cardiac muscle on the coronary vasculature depends on the (changes in) muscle length but appears to be small. In systole, coronary artery inflow is impeded, or even reversed, and venous outflow… Show more

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Cited by 221 publications
(217 citation statements)
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References 441 publications
(615 reference statements)
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“…Averages over time are given for each curve in the caption. These numbers fit in the 5 to 10 % range, in accordance to the volume changes reported by Westerhof et al (2006); Yin et al (1996). For further evaluating the effect of the incompressibility constraint, we measured the local volume change of the LV.…”
supporting
confidence: 75%
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“…Averages over time are given for each curve in the caption. These numbers fit in the 5 to 10 % range, in accordance to the volume changes reported by Westerhof et al (2006); Yin et al (1996). For further evaluating the effect of the incompressibility constraint, we measured the local volume change of the LV.…”
supporting
confidence: 75%
“…13. It clearly appears that the total volume change exceeds the expected 5 to 10 % of volume change as reported in the literature (Westerhof et al (2006);Yin et al (1996)). While volunteers #1, #2, #3 and #5 show an average volume change close to 10 % range, the other four volunteers exhibit an average volume variation in the range of 14-18%.…”
Section: Data Acquisition and Databasementioning
confidence: 55%
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“…[76][77][78]93 Briefly, this process is a result of reduced substrate l-arginine or cofactor BH 4 . Under either of these conditions the flow of electrons is delivered to molecular oxygen and superoxide is formed.…”
Section: Enosmentioning
confidence: 99%
“…The possible factors that may influence blood flow in subendocardial microvessels are the following: 1) autoregulation of the microvessels; 25) 2) direct compression by the surrounding myocardium; 26,27) 3) left ventricular luminal pressure; 4) blood pressure in the irrigating large arteries; 5) blood pressure gradient between the microvessels located in the normal wall and those located in the diseased wall; 6) interstitial pressure surrounding the microvessels, which is generated by contraction of the surrounding myocardium; 28) 7) interstitial pressure gradient across the wall 28) and between the normokinetic and diseased walls; 8) tension-time index/diastolic pressuretime index; 29) 9) pressure gradient between the microvessels and the venous trees, and 10) regional difference in microvessel density, which may be altered by angiogenesis. 30) In the present study, the timing of blood filling and collapse was closely related to the contractile state of the ventricular wall in which the microvessels were located; this resulted in blood filling during diastole and collapse during systole in normokinetic-to-hypokinetic wall segments as well as blood filling during systole and collapse during diastole in akineticto-dyskinetic wall segments.…”
Section: Discussionmentioning
confidence: 99%