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Cited by 30 publications
(34 citation statements)
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References 105 publications
(128 reference statements)
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“…Emerging evidences support the existence of other mechanisms regulating senescence escape. To date, little is known about these mechanisms (12)(13)(14)(15)(16)(17)(18). Similar data generated in human epithelial cells or in other lineages are rather rare and seem to display a more complex picture of the genetic events involved in senescence escape (18).…”
Section: Introductionmentioning
confidence: 74%
See 1 more Smart Citation
“…Emerging evidences support the existence of other mechanisms regulating senescence escape. To date, little is known about these mechanisms (12)(13)(14)(15)(16)(17)(18). Similar data generated in human epithelial cells or in other lineages are rather rare and seem to display a more complex picture of the genetic events involved in senescence escape (18).…”
Section: Introductionmentioning
confidence: 74%
“…To date, little is known about these mechanisms (12)(13)(14)(15)(16)(17)(18). Similar data generated in human epithelial cells or in other lineages are rather rare and seem to display a more complex picture of the genetic events involved in senescence escape (18). As an example, poststasis human mammary epithelial cells (HMEC), which are not expressing the p16INK4A (19), enter in senescence in response to an oncogenic stress in a DNA damagep53-independent pathway (17,18).…”
Section: Introductionmentioning
confidence: 87%
“…Nevertheless, a growing body of evidence supports the involvement of other pathways in the regulation of senescence, and in particular, in that of oncogene‐induced senescence (OIS) (Bianchi‐Smiraglia & Nikiforov, 2012; Christoffersen et al., 2010; Cipriano et al., 2011; Humbert et al., 2010; Lin et al., 2010; Scurr et al., 2010). …”
Section: Introductionmentioning
confidence: 99%
“…In addition to proliferative arrest, OIS involves the activation of a variety of signaling pathways, and senescent cells are characterized by increased size, activation of β-galactosidase, chromatin aggregates enriched for H3K9me3, activated DNA damage markers, and extracellular matrixdegrading enzymes (reviewed in ref. 23).…”
Section: Cervical Cancer Cells Are Sensitive To Treatment With the Kdmentioning
confidence: 99%