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Cited by 10 publications
(6 citation statements)
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“…An inherited homozygous deficiency in hepatic glucuronyl transferase results in the Crigler-Najjar syndrome (17)(18)(19) (15). A variety of synthetic progestational steroids inhibit glucuronyl transferase in-vitro.…”
Section: Resultsmentioning
confidence: 99%
“…An inherited homozygous deficiency in hepatic glucuronyl transferase results in the Crigler-Najjar syndrome (17)(18)(19) (15). A variety of synthetic progestational steroids inhibit glucuronyl transferase in-vitro.…”
Section: Resultsmentioning
confidence: 99%
“…There is no known inherited defect in bilirubin metabolism that results in transient unconjugated hyperbilirubinemia. An inherited defect of glucuronyl transferase activity results in permanent unconjugated hyperbilirubinemia (15)(16)(17).…”
Section: Discussionmentioning
confidence: 99%
“…Approximately twenty additional patients with this syndrome have been described and in heritance of an autosomal recessive gene has been demonstrated (110)(111)(112)(113)(114)(115). Significantly reduced glucuronide formation has been demonstrated in vivo (113)(114)(115)(116) and in vitro (116)(117)(118).…”
Section: Jaundice With Predominantly Un Conjugated Bilirubin In Serummentioning
confidence: 99%
“…Approximately twenty additional patients with this syndrome have been described and in heritance of an autosomal recessive gene has been demonstrated (110)(111)(112)(113)(114)(115). Significantly reduced glucuronide formation has been demonstrated in vivo (113)(114)(115)(116) and in vitro (116)(117)(118). Studies of bilirubin-C14 metabolism reveal that UCB is eliminated by alternative pathways which include breakdown of pigment to polar, azo-negative derivatives which are excreted in bile, a small biliary excretion of UCB and transfer of UCB across the intestinal mucosa (119).…”
Section: Jaundice With Predominantly Un Conjugated Bilirubin In Serummentioning
confidence: 99%