Comparison of echocardiography methods for assessment of left ventricular shortening and wall stress

Douglas, Pamela S.; Reicher, Nathaniel; Plappert, Theodore; Muhammad, Ali; Sutton, Martin G. St. John

doi:10.1016/s0735-1097(87)80253-x

Cited by 111 publications

(42 citation statements)

References 23 publications

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“…[15][16][17] LV meridional systolic wall stress was estimated by modifying previously published methods, assuming that LV geometry is spherical and wall thickness is uniform. 18 End-systolic wall stress (kdyne/cm 2 ) = 0.334…”

Section: Bpmentioning

confidence: 99%

Effect of Obstructive Sleep Apnea on Aortic Elastic Parameters

Tanrıverdi

Evrengül

Kaftan

et al. 2006

Circ J

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This is an obvious violation of the following "Instructions to Authors": Submission of a manuscript to the Circulation Journal implies that the article is original and that no portion (including figures or tables) is under consideration elsewhere or has been previously published in any form other than as an abstract. Previous publication includes publishing as a component of symposia, proceedings, transactions, books (or chapters), articles published by invitations or reports of any kind, as well as in electronic data bases of a public nature.Therefore, we have decided to retract the paper from Circulation Journal.As Editor-in-Chief, I regret the time that peer reviewers and others spent evaluating this paper.I sincerely hope and trust that there will be no repetition of this kind in the future. Hiroaki Shimokawa, MD, PhDEditor-in-Chief Circulation Journal (Released online August 3, 2010) R E T R A C T I O Nbstructive sleep apnea (OSA) is characterized by periodic complete or partial upper airway obstruction during sleep, causing intermittent cessation of breathing (apnea) or reduction in airflow (hypopnea) despite ongoing respiratory effort. This disorder has been described for decades, but its recognition has remained a problem. OSA appears to be an independent cardiovascular risk factor; several epidemiologic studies have identified OSA syndrome as an independent risk for systemic hypertension, coronary artery disease, stroke, and cardiac arrhythmias. [1][2][3][4] There are conflicting data in the literature on the association between OSA and left ventricular (LV) hypertrophy. Although some investigators have reported that patients with OSA develop LV hypertrophy independently of hypertension, 5 others have found no difference between patients with OSA and control subjects in LV mass (LVM). 6,7 LV Circulation Journal Vol.70, June 2006 diastolic dysfunction is regarded as an early sign of myocardial disease and an important determinant of symptoms and clinical outcome in patients with cardiovascular disease. 8 It has been shown recently that impaired LV diastolic filling occurs in OSA patients with no other active pulmonary or cardiac disease. 9 Clearly, it is important to know whether OSA alone constitutes an independent risk factor for the development of LV dysfunction and hypertrophy, and thus for increased cardiovascular morbidity and mortality.Aortic elastic properties are important determinants of blood pressure (BP) and LV function. The aorta functions not only as a conduit delivering blood to the tissues but also as an important modulator of the entire cardiovascular system, buffering the intermittent pulsatile output from the heart to provide steady flow to the capillary beds. By virtue of its elastic properties, the aorta influences LV function, structure and coronary blood flow. 10 Consequently, we hypothesized that OSA affects the functional and structural properties of large arteries, contributing to impaired LV function and changes in the LV geometry.

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Section: Bpmentioning

confidence: 99%

Effect of Obstructive Sleep Apnea on Aortic Elastic Parameters

Tanrıverdi

Evrengül

Kaftan

et al. 2006

Circ J

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“…[14][15][16] LV meridional systolic wall stress was estimated by modifying previously published methods assuming that LV geometry is spherical and wall thickness is uniform. 17 The following formula was used for this measurement: end-systolic wall stress (kdyne/cm 2 ) =0.334× SBP × LVDS/[PWS × (1+ PWS/LVDS)]; where LVDS is systolic LV diameter and PWS is systolic posterior wall thickness.…”

Section: Bpmentioning

confidence: 99%

Aortic Elastic Properties in Patients With Coronary Artery Ectasia

Tüzün

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et al. 2007

Circ J

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oronary ectasia (CE) is a clinical situation which is characterized by luminal widening of the coronary arteries and the resultant decrease in the coronary blood flow. Its incidence has been reported to be between 0.3% and 4.9% in the autopsy and cardiac catheterization series. 1 Coronary atherosclerosis (50%), congenital diseases (20%), inflammatory diseases (10-20%), collagenous and connective tissue diseases are presumed to be responsible for its cause. 2 The essential histopathological finding in the diagnosis of CE is the replacement of coronary artery media layer smooth muscle cells with hyalinized collagen, as a consequence of the increased degeneration of the media layer. 3 As a result, the loss of musculo-elastic components is observed in the media. 2 Thus, progressive artery dilatation occurs. Moreover, along with this disease, the presence of extracardiac artery dilatation has been reported in the previous studies. 4,5 Arterial stiffness, which is defined as the arterial rigidity caused by the loss of elastic tissue in the artery wall, decreases the widening capacity of the artery. Many studies searching the effects of the cardiovascular risk factors on the arteries showed that, as a consequence of the structural changes in the arteries caused by these risk factors, arterial rigidity develops and arterial widening capacity is deteriorated. It has been established that as the stiffness of the large arteries such as aorta increases, cardiovascular mortality and morbidity also increase. 6 Consequently, aortic stiffness has recently been regarded as a risk factor that needs to be treated. 7 It has also been established that aortic stiffness is increased in individuals with coronary artery disease (CAD) and in those with atherosclerosis and that it is an independent predictor of CAD. 8 In the present study, we reported that there might possibly be a relationship between the increment in aortic stiffness and left ventricular (LV) diastolic dysfunction. 9 In the current study our objective was to investigate the elastic properties of the aorta in the patients with CE and the relationship between these characteristics and LV diastolic functions. Methods Study PopulationThe cases were selected from the patients who admitted to our cardiology department for coronary angiography between the years 2001 and 2004. The study population was divided into 3 groups according to the results of the coronary angiograms. A total of 105 patients were enrolled in the present study; in the first group there were 35 patients with CE, the second group consisted of 35 patients with CAD (coronary lesion causing ≥50% luminal narrowing in at least 1 coronary artery) and the third group consisted of 35 patients with normal coronary arteries. CE was defined as localized or diffuse non-obstructive lesions of the epicardial Aortic Elastic Properties in Patients WithCoronary Artery Ectasia Nurullah Tuzun, MD; Halil Tanriverdi, MD*; Harun Evrengul, MD*; Deniz Seleci Kuru, MD*; Asim Oktay Ergene, MD** Background The purpose of the present...

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“…13 Coronary sinus flow was calculated using the thermodilution technique, 14 MVO 2 by the Fick method, and LVEF by the Simpson biplane method. 15 Circumferential end-systolic wall stress (ESWS) was calculated using Mirsky's formula described by Douglas et al 16 …”

Section: Protocolmentioning

confidence: 99%

Adjunctive handgrip during dobutamine stress echocardiography: Invasive assessment of myocardial oxygen consumption in humans

Khan

Otero

Font-Cordoba

et al. 2005

Clinical Cardiology

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SummaryBackground: Adjunctive isometric exercise in the form of sustained submaximal handgrip (HG) is considered to increase the sensitivity of dobutamine stress echo (DSE) for detection of functionally significant coronary artery disease.Hypothesis: The study was undertaken to quantify invasively the impact of HG in humans on hemodynamics and myocardial oxygen consumption (MVO2) during DSE.Methods: An invasive hemodynamic evaluation was performed during DSE and with addition of adjunctive HG in 11 subjects. Coronary sinus (CS) blood flow, right-sided and systemic pressures, oxygen saturations, and transthoracic two-dimensional echocardiography were obtained at each 3-min stage and after adding HG at peak DSE. Myocardial oxygen consumption was calculated by the Fick method and circumferential end-systolic wall stress (ESWS) by Mirsky's formula.Results: At peak DSE, heart rate, left ventricular ejection fraction, CS flow, and MVO2 increased, whereas pulmonary capillary wedge pressure (PCWP) and circumferential endsystolic wall stress (ESWS) decreased from baseline. Com-

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Comparison of echocardiography methods for assessment of left ventricular shortening and wall stress

Cited by 111 publications

References 23 publications

Effect of Obstructive Sleep Apnea on Aortic Elastic Parameters

Effect of Obstructive Sleep Apnea on Aortic Elastic Parameters

Aortic Elastic Properties in Patients With Coronary Artery Ectasia

Adjunctive handgrip during dobutamine stress echocardiography: Invasive assessment of myocardial oxygen consumption in humans

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