Prostate cancer (PCa) shows a substantial clinical heterogeneity. The
existing risk classification for PCa prognosis based on clinical factors is
not sufficient. Although some biomarkers for PCa aggressiveness have been
identified, their underlying functional mechanisms are still unclear. We
previously reported a gene-gene interaction network associated with PCa
aggressiveness based on single nucleotide polymorphism (SNP)-SNP
interactions in the angiogenesis pathway. The goal of this study is to
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