Clinical Note on Leukoderma Acquisitum Centrifugum (Sutton)

Stokes, John H.

doi:10.1001/archderm.1923.02360110040007

Cited by 11 publications

(2 citation statements)

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“…He introduced his cases of abnormal pigmentation with a review of the cellular origins of pigmentation and vitiligo. It was only later that John H. Stokes made the correlation of leukoderma acquisitum centrifugum with melanocytic nevi [ 3 ].…”

Section: Introductionmentioning

confidence: 99%

Current Challenges in Deciphering Sutton Nevi—Literature Review and Personal Experience

Nedelcu

Dobre

Brînzea

et al. 2021

JPM

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Halo nevi, known as leukoderma acquisitum centrifugum, Sutton nevus, leukopigmentary nevus, perinevoid vitiligo, or perinevoid leukoderma, together with vitiligo and melanoma-associated hypopigmentation, belong to the group of dermatoses designated as immunological leukodermas. The etiology and pathogenesis of halo nevi has not been fully elucidated. There are several mechanisms through which a lymphocytic infiltrate can induce tumoral regression. In this review, we aimed to update the knowledge about Sutton nevi starting with the clinical appearance and dermoscopic features, continuing with information regarding conventional microscopy, immunohistochemistry, and the immunological mechanisms responsible for the occurrence of halo nevi. We also included in the article original unpublished results when discussing dermoscopic, pathologic and immunohistochemical results in halo nevi. Sutton nevi are valuable models for studying antitumor reactions that the human body can generate. The slow and effective mechanism against a melanocytic skin tumor can teach us important lessons about both autoimmune diseases and anticancer defenses.

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Section: Introductionmentioning

confidence: 99%

Current Challenges in Deciphering Sutton Nevi—Literature Review and Personal Experience

Nedelcu

Dobre

Brînzea

et al. 2021

JPM

View full text Add to dashboard Cite

show abstract

“…It is not known whether the insert of the leukopigmentary anomaly came from the artist's imag¬ ination or whether one of his models actually showed the odd appear¬ ance that struck the artist as properly illustrative of demonism. Whatever the priority in uncovering the disease that now bears Sutton's name, several reports in quick succession after Sutton's report, by Shelmire,7 Bunch 8 and Stokes,9 immediately established that it is not excessively rare. In the foreign literature, reports have been made by Montpellier,10 Petges,11 Narducci,12 von Our interest in this subject was aroused by our encountering 2 cases in quick succession.…”

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confidence: 99%