2015
DOI: 10.1055/s-0035-1552934
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Abstract: The prognosis varied because of different etiologies of hydrops fetalis. Severe cases frequently had skin edema and high rate of asphyxia at birth and difficult resuscitation. Timely intrauterine interventions were helpful for successful resuscitation. A well-prepared resuscitation team and the effectiveness of resuscitation could correlate to increasing survival rate.

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Cited by 7 publications
(4 citation statements)
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“…9 Of the remaining cases, non-immune hydrops remained the predominant cause (84.6%), with chylothorax being at the top of the list of non-immune aetiologies. Other causes The predominance of non-immune causes for hydrops is similarly found in the Turkish review done by Takci et al, 1 which was a large retrospective review performed in recent years to investigate mortality risk factors in fetal hydrops and also in a recent Chinese retrospective study done by An X et al 10 The encouraging results of decreasing numbers of immune-related hydrops is proof of the success of widespread efforts in recent years in identifying rhesus negative mothers early in pregnancy, and the widespread and appropriate use of anti-D immunoglobulins to reduce isoimmunisation in these rhesus negative mothers. 11,12 We compared our findings with 2 studies done over the same period, namely that of Takci S et al, 1 3 and 4).…”
Section: Discussionmentioning
confidence: 59%
“…9 Of the remaining cases, non-immune hydrops remained the predominant cause (84.6%), with chylothorax being at the top of the list of non-immune aetiologies. Other causes The predominance of non-immune causes for hydrops is similarly found in the Turkish review done by Takci et al, 1 which was a large retrospective review performed in recent years to investigate mortality risk factors in fetal hydrops and also in a recent Chinese retrospective study done by An X et al 10 The encouraging results of decreasing numbers of immune-related hydrops is proof of the success of widespread efforts in recent years in identifying rhesus negative mothers early in pregnancy, and the widespread and appropriate use of anti-D immunoglobulins to reduce isoimmunisation in these rhesus negative mothers. 11,12 We compared our findings with 2 studies done over the same period, namely that of Takci S et al, 1 3 and 4).…”
Section: Discussionmentioning
confidence: 59%
“…[ 4 , 7 - 8 , 10 , 13 - 15 , 17 ] It has been reported that mortality in NIHF cases in the neonatal period is between 32% and 98%, and mortality in NIHF cases is higher than in immune HF cases. [ 4 , 7 - 17 , 20 , 22 - 24 ] In a recently published study involving HF infants including termination, spontaneous abortion, and intrauterine fetal losses, the survival rate in the neonatal period was found to be very low as 19.7%. [ 17 ] Differences in mortality rates in HF cases have been reported to vary depending on the gestational week, whether termination cases were included in the study, and whether the infants were born alive or not.…”
Section: Discussionmentioning
confidence: 99%
“…It has been reported that the underlying etiology, birth weight, and APGAR score at the 1 st and 5 th min of gestation, intubation, and chest compression in the delivery room, low serum albumin concentration, need for thoracentesis, and fluid accumulation in more than 2body cavities are significant prognostic factors in predicting prognosis in neonatal HF. [ 4 , 8 , 10 - 11 , 16 , 17 , 22 , 25 - 27 ] The prognosis also depends on the age of onset of hydropic changes and is associated with worse outcomes in cases of early-onset HF. [ 16 ] It is known that diagnosis in late gestational weeks and prognosis in delivery in late gestational weeks are better.…”
Section: Discussionmentioning
confidence: 99%
“…Hydrops fetalis was sonographically diagnosed by the presence of oedema in at least one fetal compartment (peritoneal, pleural or pericardial) as well as skin oedema or polyhydramnios, or fluid accumulation in two compartments. 14 There is no defined acceptable range for umbilical cord arterial lactate that reflects acidosis in the newborn. 15 We used 5.2 mmol/L as a marker of acidosis as this is a mandatory reportable incident under our state health policy.…”
Section: Methodsmentioning
confidence: 99%