Catalepsy

Limbic-diencephalic mechanisms of voluntary movement.

“…The status of perceptual-mnemonic abilities in animals made totally akinetic by central lesions has never been adequately studied. Ingram, Barris, Fisher, and Ranson (1936) were of the opinion that cats with lesions of the posterior hypothalamic area were "conscious" although they did not move about. Such a hypothesis is consistent with the observation that cats made akinetic by posterior hypothalamic lesions may exhibit good activation of the cortical electroencephalogram (EEC) but no concomitant behavioral arousal (Feldman & Waller, 1962;Lindsley, Schreiner, Knowles, & Magoun, 1950).…”

Limbic-diencephalic mechanisms of voluntary movement.

“…The status of perceptual-mnemonic abilities in animals made totally akinetic by central lesions has never been adequately studied. Ingram, Barris, Fisher, and Ranson (1936) were of the opinion that cats with lesions of the posterior hypothalamic area were "conscious" although they did not move about. Such a hypothesis is consistent with the observation that cats made akinetic by posterior hypothalamic lesions may exhibit good activation of the cortical electroencephalogram (EEC) but no concomitant behavioral arousal (Feldman & Waller, 1962;Lindsley, Schreiner, Knowles, & Magoun, 1950).…”

Neural control of feeding behavior.

“…In other studies, caudal hypothalamic lesions in rats and cats (Anand & Brobeck,195la) and in cats and monkeys (Anand et al,19SS) produced no change in food intake. To confuse the issue further, in one study in which caudal hypothalamic lesions eliminated spontaneous eating, it was reported that the animals chewed and swallowed voraciously if food was forced into their mouths (Ingram et al, 1936).…”

Limbic-diencephalic mechanisms of voluntary movement.

“…The status of perceptual-mnemonic abilities in animals made totally akinetic by central lesions has never been adequately studied. Ingram, Barris, Fisher, and Ranson (1936) were of the opinion that cats with lesions of the posterior hypothalamic area were "conscious" although they did not move about. Such a hypothesis is consistent with the observation that cats made akinetic by posterior hypothalamic lesions may exhibit good activation of the cortical electroencephalogram (EEC) but no concomitant behavioral arousal (Feldman & Waller, 1962;Lindsley, Schreiner, Knowles, & Magoun, 1950).…”

Neural control of feeding behavior.

“…In other studies, caudal hypothalamic lesions in rats and cats (Anand & Brobeck,195la) and in cats and monkeys (Anand et al,19SS) produced no change in food intake. To confuse the issue further, in one study in which caudal hypothalamic lesions eliminated spontaneous eating, it was reported that the animals chewed and swallowed voraciously if food was forced into their mouths (Ingram et al, 1936).…”

Limbic-diencephalic mechanisms of voluntary movement.

“…The status of perceptual-mnemonic abilities in animals made totally akinetic by central lesions has never been adequately studied. Ingram, Barris, Fisher, and Ranson (1936) were of the opinion that cats with lesions of the posterior hypothalamic area were "conscious" although they did not move about. Such a hypothesis is consistent with the observation that cats made akinetic by posterior hypothalamic lesions may exhibit good activation of the cortical electroencephalogram (EEC) but no concomitant behavioral arousal (Feldman & Waller, 1962;Lindsley, Schreiner, Knowles, & Magoun, 1950).…”

Neural control of feeding behavior.

“…In other studies, caudal hypothalamic lesions in rats and cats (Anand & Brobeck,195la) and in cats and monkeys (Anand et al,19SS) produced no change in food intake. To confuse the issue further, in one study in which caudal hypothalamic lesions eliminated spontaneous eating, it was reported that the animals chewed and swallowed voraciously if food was forced into their mouths (Ingram et al, 1936).…”