Calcification of Bruch's Membrane in Angioid Streaks With Homozygous Sickle Cell Disease

Pseudoxanthoma elasticum, ocular manifestations, complications and treatment

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Pseudoxanthoma elasticum, ocular manifestations, complications and treatment

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Immunohistochemical insights into sickle cell retinopathy

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Angioid streaks are part of a familial syndrome of dyserythropoietic anaemia (CDA III)

“…In sickle haemoglobinopathies, as well as in many other less frequently associated systemic disorders, such abnormal calcification is not common. In patients with sickle cell disease, iron deposition in Bruch's membrane has been suggested as the cause of the pathologic changes (Clarkson & Altman, 1982), but a recent report supports the concept that calcification also plays an important role in sickle cell disease ( Jampol et al, 1987). Aessopos et al (1992) studied 100 patients with betathalassaemia, major or intermediate.…”

Pseudoxanthoma elasticum, ocular manifestations, complications and treatment

“……”

Immunohistochemical insights into sickle cell retinopathy

“……”

“……”

“……”

Angioid streaks are part of a familial syndrome of dyserythropoietic anaemia (CDA III)

“…In sickle haemoglobinopathies, as well as in many other less frequently associated systemic disorders, such abnormal calcification is not common. In patients with sickle cell disease, iron deposition in Bruch's membrane has been suggested as the cause of the pathologic changes (Clarkson & Altman, 1982), but a recent report supports the concept that calcification also plays an important role in sickle cell disease ( Jampol et al, 1987). Aessopos et al (1992) studied 100 patients with betathalassaemia, major or intermediate.…”

Pseudoxanthoma elasticum, ocular manifestations, complications and treatment

“……”

Immunohistochemical insights into sickle cell retinopathy

“……”

“……”

“……”

Angioid streaks are part of a familial syndrome of dyserythropoietic anaemia (CDA III)

“…In sickle haemoglobinopathies, as well as in many other less frequently associated systemic disorders, such abnormal calcification is not common. In patients with sickle cell disease, iron deposition in Bruch's membrane has been suggested as the cause of the pathologic changes (Clarkson & Altman, 1982), but a recent report supports the concept that calcification also plays an important role in sickle cell disease ( Jampol et al, 1987). Aessopos et al (1992) studied 100 patients with betathalassaemia, major or intermediate.…”