Body building and myoglobinuria: report of three cases

Doriguzzi, C.; Palmucci, L.; Mongini, Tiziana; Arnaudo, Enrica; Bet, L.; Bresolin, Nereo

doi:10.1136/bmj.296.6625.826

Cited by 8 publications

(1 citation statement)

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“…More than 15 cases have already been described in the literature. [1][2][3][4][5] However, rhabdomyolysis localised to one muscle is rare, as only one case has been previously reported. 6 The clinical and biological features are similar to diffuse exertion induced rhabdomyolysis.…”

Section: Discussionmentioning

confidence: 99%

Exertion induced rhabdomyolysis of the long head of the triceps: Figure 1

Goubier

2002

Br J Sports Med

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The case is reported of bilateral rhabdomyolysis of the long head of the triceps following intensive exercise in a 30 year old male weightlifter. The diagnosis was based on myalgias localised to one muscle and raised levels of muscle enzymes. Magnetic resonance imaging helped to locate the site and extent of muscular involvement. Treatment consisted of complete rest and adequate intravenous perfusion to allow clearance of the clinical and biological abnormalities and prevent renal involvement.

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Section: Discussionmentioning

confidence: 99%

Exertion induced rhabdomyolysis of the long head of the triceps: Figure 1

Goubier

2002

Br J Sports Med

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Exercise intolerance and recurrent myoglobinuria as the only expression of Xp21 Becker type muscular dystrophy

et al. 1993

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A 9-year-old boy complained of exertional myalgias and described two episodes of myoglobinuria. His family history was negative for neuromuscular diseases. The findings of a neurological examination were normal. Serum creatine kinase was increased, ECG was normal, EMG showed slight "myopathic" signs. Muscle biopsy disclosed a small group of basophilic fibres as the only abnormality. Muscle glycolytic enzymes and carnitine palmitoyl transferase were normal. Immunoblotting using antidystrophin antibody demonstrated a protein with low molecular weight. Genomic DNA analysis showed a deletion of the HindIII fragments spanning from exon 45 to exon 48. Eight years after the first observation the patient has diffuse muscle hypertrophy without muscle weakness.

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