2005
DOI: 10.1007/s00280-005-0049-9 View full text |Buy / Rent full text
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Abstract: DL111-IT could inhibit the proliferation of cancer cells both in vitro and in vivo via a cell cycle regulation pathway.

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“…An additional difference among the cell lines investigated in the present study regards the presence of functional p53 protein inasmuch as LNCaP (wild‐type p53) differ from PC3 (p53 null) and DU‐145 (p53 mutant) 19. Nevertheless, we found that p53 expression/activation was not regulated by treatment of LNCaP cells with ST1959 (data not shown), confirming similar results obtained by others with ST1959‐treated HO‐8910 ovarian cancer cells 11.…”
Section: Discussionsupporting
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“…An additional difference among the cell lines investigated in the present study regards the presence of functional p53 protein inasmuch as LNCaP (wild‐type p53) differ from PC3 (p53 null) and DU‐145 (p53 mutant) 19. Nevertheless, we found that p53 expression/activation was not regulated by treatment of LNCaP cells with ST1959 (data not shown), confirming similar results obtained by others with ST1959‐treated HO‐8910 ovarian cancer cells 11.…”
Section: Discussionsupporting
“…Besides its immunomodulatory effects, mounting evidence suggests that ST1959 may exert inhibitory effects on proliferation of cancer cells. In fact, we and others found that this molecule displayed anti‐proliferative effects on murine NIH3T3 and several human and tumor cell lines 6–9, 11, 12. Furthermore, Burattini et al 10 reported that ST1959 caused apoptosis of HL60 cells and necrosis of SK‐N‐MC cells.…”
Section: Introductionmentioning
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“…Namely, 5 10 6 cells were injected into the right axilla of the BALB/c mice 28 . The mice were then randomly divided into three groups 10 mice in each group : control group 0.85 normal saline and AMC-treated or AACtreated groups AMC or AAC, 50 mg/kg BW .…”
Section: 7mentioning