Antibiotics for preterm rupture of membranes

The role of salivary progesterone and cervical length measurement in predicting risk of spontaneous preterm birth

“…This finding has confirmed the theory that progesterone levels are significantly lower in women who have preterm labor than those delivered at term [19]. These results are in agreement with other workers [20,21]. However, others have reported that the levels of plasma progesterone in women with preterm birth were identical to that of women with term birth [22].…”

The role of salivary progesterone and cervical length measurement in predicting risk of spontaneous preterm birth

“…This finding has confirmed the theory that progesterone levels are significantly lower in women who have preterm labor than those delivered at term [19]. These results are in agreement with other workers [20,21]. However, others have reported that the levels of plasma progesterone in women with preterm birth were identical to that of women with term birth [22].…”

Group B streptococcal and premature births: a narrativo review

“…In line with this optimistic perspective of preventing neonatal GBS infection, a study 64 suggests that the identification of the absence of a significant association between group B streptococcal bacteriuria and early onset disease should not be considered evidence that such bacteriuria is no longer an important indication of prophylaxis, but of successful prevention considering that 82% of women with bacteriuria received intrapartum prophylaxis. This finding is in line, for example, with the Cochrane meta-analysis 69 which showed that the administration of antibiotics after PROM was associated with a delay in delivery and a reduction in neonatal infection. It is important to mention that in this research 82% of all women with group B streptococcal bacteriuria also received intrapartum antibiotics.…”

Systemic immune inflammation indices: novel predictors for preterm premature rupture of membranes and associated complications

“…2 Although the exact pathophysiological mechanism of PPROM has not been clearly defined, it is widely recognized that inflammation plays a significant role in the rupture of fetal membranes. 3 This is consistent with the inclusion of antibiotic administration in the clinical management of PPROM. 4 It has been established that PPROM can occur with intraamniotic infection (detectable microorganisms along with high interleukin-6 (IL-6) concentrations in the amniotic fluid), sterile intra-amniotic inflammation (high IL-6 concentrations without detectable microorganisms), or without intra-amniotic inflammation (low IL-6 concentrations).…”

The role of salivary progesterone and cervical length measurement in predicting risk of spontaneous preterm birth

“…This finding has confirmed the theory that progesterone levels are significantly lower in women who have preterm labor than those delivered at term [19]. These results are in agreement with other workers [20,21]. However, others have reported that the levels of plasma progesterone in women with preterm birth were identical to that of women with term birth [22].…”

Group B streptococcal and premature births: a narrativo review

“…In line with this optimistic perspective of preventing neonatal GBS infection, a study 64 suggests that the identification of the absence of a significant association between group B streptococcal bacteriuria and early onset disease should not be considered evidence that such bacteriuria is no longer an important indication of prophylaxis, but of successful prevention considering that 82% of women with bacteriuria received intrapartum prophylaxis. This finding is in line, for example, with the Cochrane meta-analysis 69 which showed that the administration of antibiotics after PROM was associated with a delay in delivery and a reduction in neonatal infection. It is important to mention that in this research 82% of all women with group B streptococcal bacteriuria also received intrapartum antibiotics.…”

Systemic immune inflammation indices: novel predictors for preterm premature rupture of membranes and associated complications

“…2 Although the exact pathophysiological mechanism of PPROM has not been clearly defined, it is widely recognized that inflammation plays a significant role in the rupture of fetal membranes. 3 This is consistent with the inclusion of antibiotic administration in the clinical management of PPROM. 4 It has been established that PPROM can occur with intraamniotic infection (detectable microorganisms along with high interleukin-6 (IL-6) concentrations in the amniotic fluid), sterile intra-amniotic inflammation (high IL-6 concentrations without detectable microorganisms), or without intra-amniotic inflammation (low IL-6 concentrations).…”

The role of salivary progesterone and cervical length measurement in predicting risk of spontaneous preterm birth

“…This finding has confirmed the theory that progesterone levels are significantly lower in women who have preterm labor than those delivered at term [19]. These results are in agreement with other workers [20,21]. However, others have reported that the levels of plasma progesterone in women with preterm birth were identical to that of women with term birth [22].…”

Group B streptococcal and premature births: a narrativo review

“…In line with this optimistic perspective of preventing neonatal GBS infection, a study 64 suggests that the identification of the absence of a significant association between group B streptococcal bacteriuria and early onset disease should not be considered evidence that such bacteriuria is no longer an important indication of prophylaxis, but of successful prevention considering that 82% of women with bacteriuria received intrapartum prophylaxis. This finding is in line, for example, with the Cochrane meta-analysis 69 which showed that the administration of antibiotics after PROM was associated with a delay in delivery and a reduction in neonatal infection. It is important to mention that in this research 82% of all women with group B streptococcal bacteriuria also received intrapartum antibiotics.…”

Systemic immune inflammation indices: novel predictors for preterm premature rupture of membranes and associated complications

“…2 Although the exact pathophysiological mechanism of PPROM has not been clearly defined, it is widely recognized that inflammation plays a significant role in the rupture of fetal membranes. 3 This is consistent with the inclusion of antibiotic administration in the clinical management of PPROM. 4 It has been established that PPROM can occur with intraamniotic infection (detectable microorganisms along with high interleukin-6 (IL-6) concentrations in the amniotic fluid), sterile intra-amniotic inflammation (high IL-6 concentrations without detectable microorganisms), or without intra-amniotic inflammation (low IL-6 concentrations).…”