Annoyance and health reactions to odor from refineries and other industries in Carson, California 1972

Deane, Margaret; Sanders, George E.

doi:10.1016/0013-9351(78)90085-3

Cited by 15 publications

(24 citation statements)

References 4 publications

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“…Schneider and Costiloe ( 1969 ) measured olfactory thresholds Subjects who did not adapt or rarely adapted to the odor had the greatest incidence of stress recorded in their diary of daily life. Deane and Sanders ( 1978 ) exposure to odors from refineries and petrochemical plants between 50 and 655 ppb CH 3 SH Exposure to odors caused increase in percent reporting dizziness, nausea, or vomiting frequently ( based only on those who had previously reported being very much or moderately bothered by the odor ) . Lowest percent of chest pain among men occurred in the area of greatest exposure.…”

Section: Literature Summary Of Odors and Cardiovascular Responses (Table 1 )mentioning

confidence: 99%

“…A few negative studies observing no effect of odor on the cardiovascular system have also been reported. Deane and Sanders (1978 ) administered health questionnaires to residents from areas with different exposures to odors from refineries and petrochemical plants. Results showed the lowest percent of chest pain occurrence among men living in the area of greatest exposure.…”

Section: Shusterman and Dagermentioning

confidence: 99%

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Cardiovascular effects of odors

1999

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Several occupational and residential settings can expose both normal and sensitive human subjects to odors and irritants. These settings include intensive agricultural operations housing swine and poultry, cigarette-smoke-filled bars, landfills and manufacturing processes. The literature suggests that adverse sensory reactions to strong odors and irritants may lead to the release of catecholamines and stress hormones. Physiological and biochemical measurements related to cardiovascular risk, e.g., blood pressure, heart rate, high-density lipoprotein (HDL) cholesterol level and serum triglyceride level, may be altered as a result of exposure to odor and irritant-induced release of catecholamines. Further work in the form of field studies and chamber exposure protocols is required to determine whether the physiological and biochemical changes observed to date represent an increase in cardiovascular risk, or are reversible changes within the normal homeostatic range.

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Section: Literature Summary Of Odors and Cardiovascular Responses (Table 1 )mentioning

confidence: 99%

Section: Shusterman and Dagermentioning

confidence: 99%

Cardiovascular effects of odors

1999

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“…The concept is complex and may be considered as a perception, an emotion, an attitude or a mixture of these (Berglund et ai, 1987), although these concepts have not yet been evaluated individually. With this complexity in mind, it is not surprising that personal factors have been shown to influence odor annoyance, including age (Steinheider and Winneke, 1993;Zeidner and Shechter, 1988), gender (Deane and Sanders, 1978) and attitude toward pollution source, and to industry and authorities in general (Evans et ai, 1988). Other important factors include intensity (Cavalini etal., 1991), frequency (Hangartner and Wuest, 1994;Steinheider and Winneke, 1993) and quality (Winneke and Kastka, 1987) of the exposure.…”

Section: Introductionmentioning

confidence: 99%

Assessment of Odor Annoyance and Its Relationship to Stimulus Concentration and Odor Intensity

et al. 1998

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“…Residues from synthetic rubber manufacturing ( N = 856) Self-reported frequency of odour perception (“none”, “less than or equal to four times per month” and “greater than four times per month” Questionnaire : Self-reported symptoms Groups : Odour nuisances Gastrointestinal symptoms Mucus irritation General ill feeling Model : n.a. Effect estimated : PORs(95%CI) None Deane 1978 [ 50 ], USA, Cross-sectional Refineries and other petrochemical industries N = 291 Residents living in three residential areas nearby refineries and petrochemical plants Exposure areas estimated by dynamic olfactometry: High (Area I), Moderate (Area II), Low (Area III). Questionnaire : Self-reported symptoms Groups: Odour nuisances Gastrointestinal symptoms Mucus irritation General ill feeling Lower respiratory symptoms Upper respiratory symptoms Mood states Model: n.a.…”

Section: Resultsmentioning

confidence: 99%

“…Ten studies used distance from the source as proxy of odour exposure [ 26 , 30 , 31 , 39 , 40 , 43 , 45 , 47 , 50 , 51 ].…”

Section: Resultsmentioning

confidence: 99%

Industrial odour pollution and human health: a systematic review and meta-analysis

et al. 2021

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Objective To conduct a systematic review to evaluate the association between residential or occupational short- and long–term exposure to odour pollution from industrial sources and the health status of the exposed population. Methods The searches were conducted in Medline, EMBASE and Scopus in April 2021. Exposure to an environmental odour from industrial sources in population resident near the source or in workers was considered. We considered outcomes for which there was a biological plausibility, such as wheezing and asthma, cough, headache, nausea and vomiting (primary outcomes). We also included stress-related symptoms and novel outcomes (e.g. mood states). Risk of bias was evaluated using the OHAT tool. For primary outcomes, when at least 3 studies provided effect estimates by comparing exposed subjects versus not exposed, we pooled the study-specific estimates of odour-related effect using random effects models. Heterogeneity was evaluated with Higgins I2. Results Thirty studies were eligible for this review, mainly cross-sectional (n = 23). Only one study involved school-age children and two studies involved workers. Only five studies reported odour effects on objective laboratory or clinical outcomes. Animal Feeding Operations and waste were the most common industrial sources. The overall odds ratios in exposed versus not exposed population were 1.15 (95% CI 1.01 to 1.29) for headache (7 studies), 1.09 (95% CI 0.88 to 1.30) for nausea/vomiting (7 studies), and 1.27 (95% CI 1.10 to 1.44) for cough/phlegm (5 studies). Heterogeneity was a moderate concern. Overall, the body of evidence was affected by a definitely high risk of bias in exposure and outcome assessment since most studies used self-reported information. Conclusions Findings underline the public health importance of odour pollution for population living nearby industrial odour sources. The limited evidence for most outcomes supports the need for high quality epidemiological studies on the association between odour pollution and its effects on human health.

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Annoyance and health reactions to odor from refineries and other industries in Carson, California 1972

Cited by 15 publications

References 4 publications

Cardiovascular effects of odors

Cardiovascular effects of odors

Assessment of Odor Annoyance and Its Relationship to Stimulus Concentration and Odor Intensity

Industrial odour pollution and human health: a systematic review and meta-analysis

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