Aneurysmal dilatation of the coronary arteries: diagnostic patterns and clinical significance

Al-Harthi, Saad S.; Nouh, Mohamed S.; Saleh, Saber M.; Al-Nozha, Mansour M.

doi:10.1016/0167-5273(91)90094-6

Cited by 52 publications

(51 citation statements)

References 11 publications

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“…Previous studies have reported all three coronary vessels to be affected by CAE, but in almost 75% of patients an isolated artery was found to be ecstatic [25,26]. The reason for the higher RCA predisposition to CAE is not well understood.…”

Section: Discussionmentioning

confidence: 99%

Clinical and Angiographic Features of Patients with Coronary Artery Ectasia Compared with Stenotic Coronary Artery Disease

Abid¹,

Jalala²,

Gad³

et al. 2017

JCCR

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Section: Discussionmentioning

confidence: 99%

Clinical and Angiographic Features of Patients with Coronary Artery Ectasia Compared with Stenotic Coronary Artery Disease

Abid¹,

Jalala²,

Gad³

et al. 2017

JCCR

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“…CAE has a clinical spectrum ranging from stable angina pectoris to acute coronary syndrome. In addition to atherosclerotic plaque formations accompanying ectatic segments, microembolism to the distal coronary bed, thrombotic occlusion of the ectatic arteries and slow flow phenomenon may be responsible from the clinical picture (50)(51)(52) . Canga et al detected a significantly higher resistin level in subjects with a slow flow compared to those with a normal coronary blood flow (53) .…”

Section: Discussionmentioning

confidence: 99%

High Serum Resistin Levels in Coronary Artery Ectasia

Sayın¹,

Çetiner²,

Tekin³

et al. 2016

Kosuyolu Heart Journal

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Introduction: The etiological and pathogenic factors responsible for coronary artery ectasia (CAE) are unclear. Therefore, we aimed to compare subjects with and without CAE with respect to resistin levels and determine whether resistin plays a role in the aetiology or pathogenesis of CAE. Patients and Methods:This study enrolled a total of 81 subjects, of whom 42 had CAE [15 female (F), mean age 60.4 ± 9.0 years] and 39 had a normal coronary anatomy (22 F, mean age 56.2 ± 10.7 years). Using coronary artery diameters of the control group as reference, subjects having coronary artery dilatation that was at least 1.5 times larger than the normal adjacent segments were considered to have CAE. Resistin levels were measured from blood samples obtained on the day of the coronary angiography. Sonuç: KAE ve aterosklerozis ortak histopatolojik ve klinik özellikler arz etmektedir. Resistin, aterosklerozun gelişiminde ve klinik tablonun oluşumundaki yeri iyi bilinen bir polipeptid olup, aynı zamanda KAE oluşu-munda da rol alıyor olabilir. Resistinin KAE'nin oluşumunda ki yerinin daha iyi anlaşılabilmesi için büyük ölçekli çalışmalara ihtiyaç duyulmaktadır.

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“…Coronary ectasia is defined as a ≥1.5-fold diffuse or localized luminal dilatation of the coronary artery compared to the diameter of normal segments (1,2). CA detected in 0.3%-5.3% of consecutive angiographic studies (3,4).…”

Section: Discussionmentioning

confidence: 99%

Two Locations, Two Hits; Two Faces of The Sword: Co-Existence of Coronary and Cerebral Arterial Aneurysms

et al. 2011

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Aneurysmal dilatation of the coronary arteries: diagnostic patterns and clinical significance

Cited by 52 publications

References 11 publications

Clinical and Angiographic Features of Patients with Coronary Artery Ectasia Compared with Stenotic Coronary Artery Disease

Clinical and Angiographic Features of Patients with Coronary Artery Ectasia Compared with Stenotic Coronary Artery Disease

High Serum Resistin Levels in Coronary Artery Ectasia

Two Locations, Two Hits; Two Faces of The Sword: Co-Existence of Coronary and Cerebral Arterial Aneurysms

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