Anetoderma and human immunodeficiency virus infection

Ruiz-Rodriguez, R.

doi:10.1001/archderm.128.5.661

Cited by 13 publications

(6 citation statements)

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“…Primary anetoderma should be distinguished from secondary anetoderma, which consists of areas of atrophic skin that appear in the same site as a previous specific skin lesion. There are numerous and heterogeneous dermatoses associated with secondary anetoderma, including syphilis, 3 leprosy, 3 sarcoidosis, 3 granuloma annulare, 4 tuberculosis, 3 human immunodeficiency virus infection, 5,6 folliculitis, 7 angular cheilitis, 8 acrodermatitis chronica atrophicans, 3 lupus erythematosus, 3 amyloidosis, 3 lymphocytoma cutis, 9 cutaneous plasmacytoma, 9 cutaneous B‐cell lymphoma, 10 urticaria pigmentosa, 11,12 juvenile xanthogranuloma, 13 immunocytoma, 14 prurigo nodularis, 15 pilomatricomas, 16–18 sites of attachment of gel electrocardiographic electrodes in premature infants, 19–21 varicella, 22 acne vulgaris, 22 lichen planus, 22 melanocytic naevus, 22 dermatofibroma, 22 xanthoma, 22 and following penicillamine therapy 23 or hepatitis B immunization 24 . The pathogenesis of both primary and secondary anetoderma remains uncertain.…”

Section: Discussionmentioning

confidence: 99%

Anetodermic mycosis fungoides: a new clinicopathological variant of mycosis fungoides

et al. 2007

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Mycosis fungoides is the most common type of primary cutaneous T-cell lymphoma. Several rare clinicopathological variants of mycosis fungoides have been described. Patients with these variants often also have classic mycosis fungoides at other sites of the body. Anetoderma is a cutaneous disorder in which multiple, oval lesions or atrophic plaques with wrinkled surface develop progressively due to loss of the dermal elastic tissue. Primary anetoderma occurs when there is no underlying associated disease and it arises on clinically normal skin, whereas secondary anetoderma appears in the same site as a previous specific skin lesion. There is a large list of heterogeneous dermatoses associated with secondary anetoderma. Two patients developed areas of secondary anetoderma on plaque stage lesions of mycosis fungoides. The lesions consisted of exophytic nodular lesions, with very soft consistency on palpation, scattered over the hyperpigmented plaques in one patient and violaceous indurated plaques with overlying epidermal atrophy and mild scale in the other. Histopathological study demonstrated that the cells involving the dermis were mainly T-helper lymphocytes, with few histiocytes and some multinucleate giant cells engulfing distorted elastic fibres. Elastic tissue stain demonstrated that elastic fibres were almost completely absent in the dermis of the anetodermic lesions. Anetodermic mycosis fungoides should be added to the list of clinicopathological variants of mycosis fungoides and mycosis fungoides should also be considered as a possible disease causing secondary anetoderma. Anetodermic mycosis fungoides shows clinical and histopathological features different from those of granulomatous slack skin.

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Section: Discussionmentioning

confidence: 99%

Anetodermic mycosis fungoides: a new clinicopathological variant of mycosis fungoides

et al. 2007

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“…Anetoderma seems to be more frequent in central Europe than elsewhere, suggesting a possible relationship to chronic atrophic acrodermatitis due to Borretia in some cases (7). Recently the development of anetoderma in an early course of HIV infection has been reported in three patients (12). The authors suggested that anetoderma may be an early manifestation of HIV disease, perhaps autoimmune in its pathogenesis.…”

Section: Discussionmentioning

confidence: 99%

Primary Anetoderma in Children: Report of Two Cases and Literature Review

Karrer

Szeimies

Stolz

et al. 1996

Pediatric Dermatology

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Two boys, age 7 and 9 years, with the diagnosis of primary anetoderma are presented. In one patient a growing number of indolent lesions developed for one year on the neck. The other boy complained of single lesions appearing over four months on the arms, feet, and chest wall. Individual lesions measured up to 1 cm in diameter and showed a palpable herniation phenomenon and wrinkled surface. The lesions did not have an inflammatory onset. Histologically, in both patients the diagnosis of anetoderma was verified by loss of elastic fibers and a lympho-histiocytic infiltration in the middermis. Administration of oral penicillin for three weeks did not result in marked improvement, and atrophic macules continued to appear in the younger boy. Although no autoimmunologic abnormalities or other associated diseases have arisen in our patients, long-term follow-up is mandatory to detect autoimmune disorders that are reported to occur in the course of the disease.

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“…Numerous infectious diseases have also been linked with anetoderma (tuberculosis, syphilis, etc.). Most recently, patients who are HIV‐1 positive have been found to develop secondary anetoderma 9,10 . The skin lesions in patients with HIV are believed to form as a result of immune dysregulation rather than of the viral infection itself.…”

Section: Discussionmentioning

confidence: 99%

Familial anetoderma

et al. 2003

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A 31-year-old Caucasian male presented with a history of erythematous, saccular outpouchings of the skin on his back, shoulders, and upper extremities (Fig. 1). The patient reported that his mother and aunt had a similar skin disorder, which initially began with inflammation, but healed leaving lax skin in its wake. He did not recall the name of the skin condition. Physical examination revealed large confluent zones of sac-like protrusions of erythematous skin on the back and shoulders. Histopathologic examination of the excisional biopsy revealed a relatively unremarkable epidermis. Perivascular lymphocytes were present in small numbers in the papillary dermis. Adnexal structures and deep dermis remained intact. The acid-orcein-Giemsa stain highlighted the absence of elastic fibers within the mid and lower reticular dermis (Fig. 2a,b), consistent with anetoderma.

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Anetoderma and human immunodeficiency virus infection

Cited by 13 publications

References 0 publications

Anetodermic mycosis fungoides: a new clinicopathological variant of mycosis fungoides

Anetodermic mycosis fungoides: a new clinicopathological variant of mycosis fungoides

Primary Anetoderma in Children: Report of Two Cases and Literature Review

Familial anetoderma

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