2000
DOI: 10.1002/1097-0185(20000701)259:3<::aid-ar10>3.3.co;2-7 View full text |Buy / Rent full text
|
|

Abstract: This essay lays the groundwork for the concept that "anatomy" in the new millennium will be a subject that is increasingly based on understanding the parallel relationships between systems of genes on chromosomes and the structures defined by these genes. The concept of Anatomic Genomics is introduced in terms of systems of genes on chromosomes, which actually mirror the biology of anatomically defined systems. A case is made for the possibility that genomes may be structured in ways that make local but not ne… Show more

Help me understand this report

Search citation statements

Order By: Relevance
Select...
1
1
0
7
0

Year Published

2005
2005
2011
2011

Publication Types

Select...
5

Relationship

3
2

Authors

Journals

0
7
0
Order By: Relevance
“…(CFTR) gene (1)(2)(3)(4). CFTR mutations, of which the most common is ⌬F508-CFTR, cause a massive proinflammatory phenotype in the lung, which manifests in the airway by high levels of IL-8 and other proinflammatory cytokines and chemokines (5)(6)(7).…”
mentioning
Create an account to read the remaining citation statements from this report. You will also get access to:
  • Search over 1.2b+ citation statments to see what is being said about any topic in the research literature
  • Advanced Search to find publications that support or contrast your research
  • Citation reports and visualizations to easily see what publications are saying about each other
  • Browser extension to see Smart Citations wherever you read research
  • Dashboards to evaluate and keep track of groups of publications
  • Alerts to stay on top of citations as they happen
  • Automated reference checks to make sure you are citing reliable research in your manuscripts
  • 7 day free preview of our premium features.

Trusted by researchers and organizations around the world

Over 130,000 students researchers, and industry experts at use scite

See what students are saying

rupbmjkragerfmgwileyiopcupepmcmbcthiemesagefrontiersapsiucrarxivemeralduhksmucshluniversity-of-gavle
“…(CFTR) gene (1)(2)(3)(4). CFTR mutations, of which the most common is ⌬F508-CFTR, cause a massive proinflammatory phenotype in the lung, which manifests in the airway by high levels of IL-8 and other proinflammatory cytokines and chemokines (5)(6)(7).…”
mentioning
“…cytokines; chemokines; inflammation; gene regulation CYSTIC FIBROSIS (CF) is the most common fatal autosomal recessive disease in the U.S. and Europe, and is due to mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) gene (5,9,11,15). CF is characterized by a massive proinflammatory phenotype in the lung, due to the secretion of high levels of IL-8 and other proinflammatory cytokines and chemokines (1,4,6,17,18).…”
mentioning
“…CYSTIC FIBROSIS (CF) is the most common life-limiting autosomal recessive disease in the United States and Europe and is due to mutations in the CFTR gene (10,20,26,30). CFTR mutations, of which the most common is [⌬F508]CFTR, cause a massive proinflammatory phenotype in the lung, which is chemically manifest in the airway by high levels of IL-8 and other proinflammatory cytokines and chemokines (1,15,31).…”
mentioning