An epidemiologic assessment of primary endocardial fibroelastosis

Mitchell, Shiela C.; Froehlich, Luz A.; Banas, John S.; Gilkeson, Mary R.

doi:10.1016/0002-9149(66)90431-0

Cited by 26 publications

(3 citation statements)

References 53 publications

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“…Primary endocardial fibroelastosis in children is diagnosed when there are no valvular heart lesions, no inflammation, or no exactly defined myocardial degeneration (Mitchell, Froehlich, Banas, and Gilkeson, 1966). The results of our own investigations, as also those of otherauthors (Andersen andKelly, 1956;Kelly and Andersen, 1956;Boj and Pikiel, 1964), show that even in the so-called primary endocardial fibroelastosis the endocardial changes are secondary to the myocardial damage.…”

Section: Discussionsupporting

confidence: 65%

Endocardial fibroelastosis in children with special reference to the lesions of cardiac ganglia

Zółtowska

1971

J Clin Pathol

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SYNOPSIS In all cases of primary endocardial fibroelastosis investigated diffuse degenerative changes in the ganglion cells were detected. Thickened endocardium was formed by the proliferation of mesenchymal cells located under the endothelial endocardium and of the mesenchyme surrounding the necrobiotic muscle cells. In some cases 'axial' vacuolar degeneration, necrobiosis, and necrosis of muscle cells, interstitial oedema, and dilatation of capillaries and lymphatic spaces were found in the whole myocardium. In all cases these changes were in the subendocardial layer.In the cases discussed morphological changes in the endocardium were seen to be secondary to lesions in myocardial and possibly in neural structures.

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Section: Discussionsupporting

confidence: 65%

Endocardial fibroelastosis in children with special reference to the lesions of cardiac ganglia

Zółtowska

1971

J Clin Pathol

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“…as a congenital abnormality of the heart. occurs in about one case out of 5000 to 6000 births (Mitchell et al. 1966).…”

Section: Ntroductionmentioning

confidence: 99%

Prenatal diagnosis of endocardial fibroelastosis

Bovicelli¹,

Picchio²,

Pilu³

et al. 1984

Prenatal Diagnosis

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In a case of fetal heart failure caused by endocardial fibroelastosis, prenatal echocardiography clearly demonstrated a thickened endocardium. We therefore suggest that an abnormal endocardium may be detected in utero by ultrasound, thus representing an important clue in the differential diagnosis of fetal nonimmune hydrops and in the evaluation of pregnancies at risk for endocardial fibroelastosis.

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“…There is no other heart disease in the family. Mitchell et al (1966) laid down stringent criteria for the diagnosis of endocardial fibroelastosis. The disease can be diagnosed with reasonable certainty clinically, and cardiac catheterization and angiocardiography give characteristic findings.…”

Section: Case Materialsmentioning

confidence: 99%