Alteraciones gingivales no relacionadas con placa

Palacios-Sánchez, Begoña; Cerero-Lapiedra, Rocío; Campo‐Trapero, Julián; Esparza-Gómez, Germán

doi:10.4321/s1138-123x2006000100003

Cited by 4 publications

(4 citation statements)

References 11 publications

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“…The histopathological findings are consistent with most bibliographic findings, which report a stratified squamous epithelium often hyperplastic and keratinized, which can display inflammatory changes such as exocytosis, acanthosis and proliferation of epithelial cones 17 . This case also exhibited parakeratinized stratified squamous epithelium with areas of acanthosis and atrophy.…”

Section: Discussionsupporting

confidence: 88%

Inflammatory fibrous hyperplasia in pediatric dentistry due to deleterious habit: case report

Danelon¹,

Emerenciano²,

Silva³

et al. 2020

Arch Health Invest

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This paper reports the clinical case of a pediatric dentistry patient, presenting a correlation between non-nutritive suction and inflammatory fibrous hyperplasia. A 10-year-old female patient attended the Pediatric Dentistry Clinic of the Araçatuba Dental School-Brazil (FOA/UNESP) with her mother, who reported a "little ball under the upper lip" of the daughter, observed three months before, with gradual increase. The clinical examination revealed poor positioning of tooth 21 and a fibrous hyperplasia at 2 cm from the upper region of the respective tooth. A presumptive diagnosis of inflammatory fibrous hyperplasia was established. The proposed treatment was lesion excision, fabrication of a Hawley appliance for repositioning of tooth 21 and counseling on the need to cease the deleterious digital sucking habit. We conclude that elimination of the traumatic agent is fundamental for therapeutic success, in addition to surgical removal.Descriptors: Hyperplasia; Fingersucking; Habits.ReferencesEspinoza-Zapata M, Loza-Hernández G, Mondragón-Ballesteros R. Prevalence of buccal mucosa lesions in pediatric patients. Preliminary report. Cir Cir. 2006; 74(3):153-57.Cawson R, Eveson J. Oral pathology and diagnosis: Color atlas with integrated text. Phipadelphia, USA: Saunders, 1995;1-128.Rodriguez AF, Sacsaquispe SJ. Hyperplasia Fibrosa Inflamatória y posibles factores asociados em adultos mayores. Rev Estomatol Heridiana.2005;15(2):139-44.American Academy of Pediatric Dentistry. Management considerations for pediatric oral surgery and oral pathology. Pediatr Dent 2018;40(6):373-82.Neville B, Damm DD, Allen C, Chi A. Oral and Maxillofacial Pathology, 4th ed. Elsevier; 2016.Góis EG, Ribeiro HC, Vale MP, Paiva SM, Serra-Negra JMC, Ramos-Jorge ML et al. Influence of nonnutritive sucking habits, breathing pattern and adenoid size on the development of malocclusion. Angle Orthod. 2008;78(4):647-54.Scarpelli BB, Berger SB, Punhagui MF, Oliveira CAZ, Ferelle A, Oltramari-Navarro PVP. Evaluation of a preventive educational program for malocclusions: 7-year study. Braz oral res. 2016;30(1):e119.Cavalvanti AL, Bezerra PKM, Moura C, Bezerra PM, Granville-Gracia AF. Relationship between malocclusion and deleterious oral habits in preschool children in Campina Grande, PB, Brazil. Stom Glass S. 2008;55:154-62.Montaldo L, Montaldo P, Cuccaro P, Caramico N, Minervini G. Effects of feeding on non-nutritive sucking habits and implications on occlusion in mixed dentition. Int J Paediatr Dent. 2011;21(1):68-73.Vasconcelos FMN, Massoni ACLT, Heimer MV, Ferreira AMB, Katz CRT, Rosenblatt A. Non-nutritive sucking habits, anterior open bite and associated factors in Brazilian children aged 30-59 months. Braz Dent J. 2011;22(2):140-45.Suhani RD, Suhani MF, Muntean A, Mesaros M, Badea ME. Deleterious oral habits in children with hearing impairment. Clujul Med. 2015;88(3):403-7.Bueno SB, Bittar TO, Vazquez FL, Meneghim MC, Pereira AC. Association of breastfeeding, pacifier use, breathing pattern and malocclusions in preschoolers. Dental Press J Orthod.2013;18(1):30e1-6.Gheno JN, Martins MAT, Munerato MC, Hugo FN, Sant'ana Filho M, Weissheimer C et al. Oral mucosal lesions and their association with sociodemographic, behavioral, and health status factors. Braz oral res. 2015;29(1):00289.Warren JJ, Bishara SE, Steinbock KL, Yonezu T, Nowak AJ. Effects of oral habit´s duration on dental characteristics in the primary dentition. J Am Dent Assoc. 2001;132(12):1685-93.Barros RMG, Campos KSM, Cabral LM. Relato de caso clínico de hiperplasia fibrosa inflamatória. Rev Odontol Araçatuba. 2014;35(2):15-18.Dutra KL, Longo L, Grando LJ, Riveiro ERC. Incidence of reactive hyperplastic lesions in the oral cavity: a 10 years’ retrospective study in Santa Catarina, Brazil. Braz J Otorhinolaryngol 2019; Braz j otorhinolaryngol. 2019;85(4):399-407.Palacios-Sánchez B, Cerero-Lapiedra R, Campo-Trapero J, Esparza-Gómez GC. Alteraciones gingivales no relacionadas con placa. RCOE. 2006;11(1):43-55.Coelho CM, Sousa YT, Daré AM. Denture-related oral mucosal lesions in a Brazilian school of dentistry. J Oral Rehabil. 2004;31(2):135-39.

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Section: Discussionsupporting

confidence: 88%

Inflammatory fibrous hyperplasia in pediatric dentistry due to deleterious habit: case report

Danelon¹,

Emerenciano²,

Silva³

et al. 2020

Arch Health Invest

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“…Los casos que presentan sintomatología están asociados a ulceración concomitante de la lesión; se muestra eritematoso si está ulcerado, semejando un Granuloma Piógeno. La consistencia de la lesión es variada de firme a flácida; puede presentarse con una superficie lisa, con base sésil y ocasionalmente pediculada; sangra fácilmente a la palpación o al cepillado (9) . Histológicamente se caracteriza por una sobreproducción de tejido conjuntivo fibroso, rico en fibras de colágeno y fibroblastos, delimitado por epitelio escamoso superficial; existe presencia de infiltrado inflamatorio crónico (linfocitos y células plasmáticas, principalmente y ocasionalmente leucocitos polimorfonucleares pueden estar presentes) y pocos vasos sanguíneos.…”

Section: Figuraunclassified

Hiperplasia Fibrosa Inflamatoria: reporte de un caso

Romero¹,

Quiroz²,

Torres³

et al. 2011

Rev. Clin. Periodoncia Implantol. Rehabil. Oral

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RESUMENIntroducción: La Hiperplasia Fibrosa Inflamatoria es una alteración caracterizada por un agrandamiento del tejido conectivo; se considera una lesión reactiva ya que se presenta como respuesta a una irritante crónico local; es una lesión común en pacientes adolescentes y de edad adulta debido a que su etiología principal se asocia a maloclusión, al uso de aparatología protésica u ortodóntica mal ajustada, así como a la presencia de biofilm. Sin embargo, la población infantil, principalmente en dentición mixta, no está exenta de presentar esta alteración, aunque en menor proporción. Objetivo: Presentar el caso clínico de un paciente en dentición mixta que presenta una Hiperplasia Fibrosa Inflamatoria con el fin de presentar las características clínicas e histopatológicas de la lesión, enfatizando la importancia de un temprano y correcto diagnóstico. Presentación del Caso: Paciente masculino de 8 años de edad que se presenta a la clínica por presentar lesiones cariosas y malposición dental. A la exploración se observa un agrandamiento gingival localizado, en el órgano dentario 41. Se decide tomar una biopsia de la lesión para establecer el diagnóstico. Conclusión: Debido a que existen varias lesiones con características clínicas muy similares a las que presentaba el paciente, es necesario conocer los diagnósticos diferenciales, así como realizar el diagnóstico temprano de la lesión para poder llevar a cabo un tratamiento eficaz y adecuado. Rev. Clin. Periodoncia Implantol. Rehabil. Oral Vol. 4(2); 74-79, 2011.Palabras clave: Hiperplasia, biofilm, maloclusión dental, agrandamiento gingival. ABSTRACT Introduction:The Inflammatory Fibrous Hyperplasia (IFH) is a disorder consisting in a connective tissue overgrowth; it is considered a reactive lesion because it appears in response to a chronic and local irritant; this is a very common oral lesion in adolescents and adults, its etiology is associated with dental malocclusion, use of over-extended prosthetic and orthodontic appliances, and with the presence of biofilm (plaque). Nevertheless, child population, mainly patients in mixed dentition, are not exempt from presenting this lesion, in a lower proportion, though. Objective: The aim of this case report is to present the clinical case of a patient in mixed dentition, who displays an Inflammatory Fibrous Hyperplasia, with the purpose of showing the clinical and histopathological features of this lesion, emphasizing the importance of an early and correct diagnosis. Case Report: A 8 year-old male patient, was referred to the Pediatric Dentistry Clinic of the Division of Post-graduate studies and Research, of the School of Dentistry, UNAM, for showing decay lesions and dental malposition. Clinical examination revealed a localized gingival over-growth associated to the teeth 41. It was decided to take a biopsy of the lesion to establish the final diagnosis. Conclusion: Because there are numerous lesions with pretty similar clinical features to the ones the patient presented, it is imperative and essential to know th...

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“…Estas hormonas provocan alteraciones vasculares, celulares, microbiológicas e inmunológicas. Sin embargo, en ausencia de placa estos cambios no dan lugar a enfermedad periodontal (2).…”

Section: Introductionunclassified

“…Palacios Sánchez y cols. realizaron una revisión bibliográfica donde determinaron que el 5% de las gestantes presentan un granuloma piógeno durante el embarazo, éste suele aparecer en el primer o segundo mes y remitir tras el parto (2).…”

Section: Introductionunclassified