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“…In the setting of acrolein-induced lung injury, Lu et al (14) demonstrated that Alda-1 treatment promoted the detoxification of acrolein and further improved lung vascular endothelial barrier dysfunction in mice. In the setting of hypoxia-induced lung injury, Patil et al (15,16) demonstrated that Alda-1 treatment improved mitochondrial membrane potential and decreased oxidative stress and apoptosis in human lung vascular endothelial cells, and alleviated lung inflammation and oxidative stress and enhanced its cell survival via the Akt and mammalian target of rapamycin pathways in mice. In the setting of sepsis-induced lung injury, Cao et al (17) demonstrated that Alda-1 treatment simultaneously reduced lung pyroptosis and ferroptosis and therefore alleviated its injury in mice.…”
Section: Discussionmentioning
confidence: 99%
“…In the setting of acrolein-induced lung injury, Lu et al (14) demonstrated that Alda-1 treatment promoted the detoxification of acrolein and further improved lung vascular endothelial barrier dysfunction in mice. In the setting of hypoxia-induced lung injury, Patil et al (15,16) demonstrated that Alda-1 treatment improved mitochondrial membrane potential and decreased oxidative stress and apoptosis in human lung vascular endothelial cells, and alleviated lung inflammation and oxidative stress and enhanced its cell survival via the Akt and mammalian target of rapamycin pathways in mice. In the setting of sepsis-induced lung injury, Cao et al (17) demonstrated that Alda-1 treatment simultaneously reduced lung pyroptosis and ferroptosis and therefore alleviated its injury in mice.…”
Section: Discussionmentioning
confidence: 99%