1998
DOI: 10.1007/s004310050767
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Acute pulmonary oedema due to transient myocardial dysfunction: an uncommon cause of respiratory distress in the term neonate

Abstract: Pulmonary oedema due to transient myocardial dysfunction can be a rare cause in the differential diagnosis of respiratory distress of the newborn. Myocardial impairment after probable hypoxia can be present without concomitant encephalopathy.

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Cited by 8 publications
(3 citation statements)
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References 14 publications
(18 reference statements)
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“…Infants with high urine output may thrive despite low eGFR [25]. In infants, eGFR is a weaker determinant for initiating dialysis.…”
Section: Introductionmentioning
confidence: 99%
“…Infants with high urine output may thrive despite low eGFR [25]. In infants, eGFR is a weaker determinant for initiating dialysis.…”
Section: Introductionmentioning
confidence: 99%
“…However, the pathogenesis of TTN is largely unknown. Mild degree of pulmonary immaturity (3), slight and transient surfactant deficiency (4) or myocardial left‐heart failure due to asphyxia, resulting in pulmonal interstitial edema (5) are discussed to be underlying pathophysiological factors in the etiology of TTN. Surfactant‐associated protein B (SP‐B) was demonstrated to be an important component of the pulmonal surfactant system for the immediate postnatal pulmonary adaptation (6).…”
Section: Introductionmentioning
confidence: 99%
“…A further hypothesis is TTN to result from a slight and transient surfactant deficiency in affected infants [20]. Myocardial leftheart failure due to asphyxia resulting in pulmonanary interstitial edema is discussed as pathological mechanism in the cause of TTN [22].…”
Section: Introductionmentioning
confidence: 99%