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Cited by 10 publications
(2 citation statements)
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“…It is a group of genetic changes characterized by the dominance of hemoglobin (Hb) S, including sickle cell anemia and double heterozygosity, that is, associations between Hb S and other hemoglobin variants, such as Hb D and Hb C, and interactions with thalassemia (Hb S/α thalassemia, Hb S/β + thalassemia, and Hb S/β 0 thalassemia) 2 . Sickle cell anemia is not only the most clinically and hematologically severe genetic variation but also the most prevalent one 3 . Hb S is characterized by a point mutation occurring at Position 6 of the β chain of globin, where the glutamic acid (amino acid) is replaced with valine 4 .…”
Section: Introductionmentioning
confidence: 99%
See 1 more Smart Citation
“…It is a group of genetic changes characterized by the dominance of hemoglobin (Hb) S, including sickle cell anemia and double heterozygosity, that is, associations between Hb S and other hemoglobin variants, such as Hb D and Hb C, and interactions with thalassemia (Hb S/α thalassemia, Hb S/β + thalassemia, and Hb S/β 0 thalassemia) 2 . Sickle cell anemia is not only the most clinically and hematologically severe genetic variation but also the most prevalent one 3 . Hb S is characterized by a point mutation occurring at Position 6 of the β chain of globin, where the glutamic acid (amino acid) is replaced with valine 4 .…”
Section: Introductionmentioning
confidence: 99%
“…2 Sickle cell anemia is not only the most clinically and hematologically severe genetic variation but also the most prevalent one. 3 Hb S is characterized by a point mutation occurring at Position 6 of the β chain of globin, where the glutamic acid (amino acid) is replaced with valine. 4 This modified Hb polymerizes in its deoxygenated state and at high concentrations, resulting in abnormally rigid, nondeformable red blood cells (sickle red blood cells).…”
Section: Introductionmentioning
confidence: 99%