“…4,5 Recent evidence shows that direct myocardial injury by the virus is less relevant than indirect lesions from systemic inflammation and hypercoagulability in these patients. 6,7 Despite understanding these pathogenic mechanisms, the prevalence of myocardial involvement in COVID-19 remains debatable. Diagnosis of myocardial injury based exclusively on the elevation of serum biomarkers, such as troponin, may overestimate the number of cases.…”