Cervical radiculopathy secondary to a tortuous vertebral artery

Horgan, Michael A.; Hsu, Frank P.K.; Frank, Edmund

doi:10.3171/jns.1998.89.3.0489

Cited by 25 publications

(13 citation statements)

References 4 publications

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“…The presence of such structures along the course of a nerve is necessary for the development of an entrapment neuropathy (Bennett, 1985;Fisher and Gorelick, 1985;Pratt, 1986;Ravindran, 2003). The supraclavicular nerves may be injured at different points along their course (Mehta and Birch, 1997;Detwiler et al, 1998;Horgan et al, 1998). The specific conditions for entrapment of these nerves, however, exist in the region of the clavicle.…”

Section: Variant Muscular Structuresmentioning

confidence: 99%

Study of variant anatomical structures (bony canals, fibrous bands, and muscles) in relation to potential supraclavicular nerve entrapment

Jelev

Surchev

2006

Clinical Anatomy

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Since the definition of supraclavicular nerve entrapment syndrome by Gelberman et al. (Gelberman et al. [1975] J. Bone Joint Surg. Am. 57:119) a number of clinical cases of this specific entrapment neuropathy have been reported. In all these cases, the nerve injury has been attributed to the location of the supraclavicular nerve branch in a narrow canal in the clavicle. However, in the anatomical literature, variations in the course of the supraclavicular nerves have not only been ascribed to bony canals but also to abnormal fibrous and muscular structures. Considering the fact that the existence of a narrow site with rigid walls along the course of a nerve is essential for the development of an entrapment neuropathy, our study examines all the variant anatomical structures with a possible role in supraclavicular nerve entrapment. We describe three groups of anatomical structures with close relation to the course of the supraclavicular nerves-transclavicular canals, fibrous bands, and unusual muscular structures. Based on the characteristics of the variations found, for the first time, we suggest that in addition to the bony canals through the clavicle certain fibrous and muscular structures could also be an anatomical basis for supraclavicular nerve entrapment syndrome.

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Section: Variant Muscular Structuresmentioning

confidence: 99%

Study of variant anatomical structures (bony canals, fibrous bands, and muscles) in relation to potential supraclavicular nerve entrapment

Jelev

Surchev

2006

Clinical Anatomy

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“…4 Horgan et al elected to conservatively treat their patient who presented with cervical radiculopathy secondary to a right VA loop at the C4/5 level. 5 Our patient had complex left-sided VA loops at the C2/3 and C4 levels. His symptoms were clearly related to cervical motion, especially during extension and flexion.…”

Section: Discussionmentioning

confidence: 95%

“…[14][15][16] The presence of isolated radiculopathy could be explained by direct compression of the nerve root by the VA loop. 5 Sakaida et al reported a 62-year-old man who presented with a combination of vertebrobasilar insufficiency and left-sided C5 radiculopathy due to a VA loop that migrated into a widened left C4/5 intervertebral foramen. The patient underwent vascular reconstruction of the VA loop through an anterolateral approach after a balloon occlusion test.…”

Section: Discussionmentioning

confidence: 98%

Multiple symptomatic vertebral artery loops treated with posterior cervical fusion

Dahdaleh

Albert

Hasan

2010

Journal of Clinical Neuroscience

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“…22) Less common causes include trauma, intra-or extraspinal tumors, synovial cysts, 15,20) synovial chondromatosis, 4,13) dural arteriovenous fistulae, 11) and tortuous vertebral arteries. 8) None of these conditions appeared to be causative in our patient. Hypertrophic synovial mass is an extremely rare cause with only 2 reported cases.…”

Section: Introductionmentioning

confidence: 85%

Hypertrophic Synovial Mass Resulting in C8 Radiculopathy -Case Report-

Sasamori

Isu

Morimoto

et al. 2010

Neurol. Med. Chir.(Tokyo)

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A 58-year-old man presented with a hypertrophic synovial mass manifesting as left C8 radiculopathy. Computed tomography revealed hypertrophic changes in the left C7-T1 facet joint and widening of the interarticular space. Magnetic resonance imaging showed left C7-T1 foraminal stenosis attributable to a periarticular mass enhanced with gadolinium. Decompression surgery of the left C8 nerve root was performed. During left C7-T1 facetectomy we detected inflamed soft tissue occupying the interarticular space with extension toward the intervertebral foramen. The mass markedly compressed the left C8 nerve root and was gross totally removed. The histological diagnosis was proliferated fibrous connective tissue compatible with hypertrophic synovial tissue. Postoperatively, his neurological condition improved remarkably. The surgical and pathological findings in our patient indicate that this clinical condition is important in the differential diagnosis in adult patients presenting with cervical radiculopathy.

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Cervical radiculopathy secondary to a tortuous vertebral artery

Cited by 25 publications

References 4 publications

Study of variant anatomical structures (bony canals, fibrous bands, and muscles) in relation to potential supraclavicular nerve entrapment

Study of variant anatomical structures (bony canals, fibrous bands, and muscles) in relation to potential supraclavicular nerve entrapment

Multiple symptomatic vertebral artery loops treated with posterior cervical fusion

Hypertrophic Synovial Mass Resulting in C8 Radiculopathy -Case Report-

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