2006
DOI: 10.1590/s1415-52732006000500008
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Abstract: A B S T R A C TThe liver is a strategic organ in the metabolism of macro and micronutrients; when its functioning is compromised, it may cause some change in the nutritional status of vitamin A. The purpose of this article is to review scientific evidence in literature on the liver metabolism of vitamin A, the role of ethanol and retinol interactions on hepatic morphology, besides the alterations in the metabolism of this vitamin in alcoholic liver disease. Data were collected from Medline database. The liver … Show more

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Cited by 2 publications
(2 citation statements)
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“…Hepatic cells involvement would therefore limit the metabolic functions of the liver. These results are supported by Paula et al (2006) [32] , Di Sario et al (2007) [3] , Nair (2010) [33] , Tanumihardjo (2011) [34] , Chaves et al (2015) [5] and Konstantakis et al (2016) [35] who showed that the serum rates of vitamins A, D and E were decreased during liver diseases. The pretreatment of CCl 4 -intoxicated rats led to increase serum concentrations of vitamins A, D and E. These results are corroborated by Premalatha and Parameswari (2013) [36] who indicated that chrysin increased the serum concentrations of vitamins A and E in streptozotocin-intoxicated rats.…”
Section: Discussionsupporting
confidence: 53%
“…Hepatic cells involvement would therefore limit the metabolic functions of the liver. These results are supported by Paula et al (2006) [32] , Di Sario et al (2007) [3] , Nair (2010) [33] , Tanumihardjo (2011) [34] , Chaves et al (2015) [5] and Konstantakis et al (2016) [35] who showed that the serum rates of vitamins A, D and E were decreased during liver diseases. The pretreatment of CCl 4 -intoxicated rats led to increase serum concentrations of vitamins A, D and E. These results are corroborated by Premalatha and Parameswari (2013) [36] who indicated that chrysin increased the serum concentrations of vitamins A and E in streptozotocin-intoxicated rats.…”
Section: Discussionsupporting
confidence: 53%
“…Larger studies considering long-term outcomes in those patients are still lacking. If supplementation is considered, one must remember that because of the impaired hepatic metabolism in ethanol-induced liver disease, and because of potentiating the retinol toxicity by ethanol, the usual doses of vitamin A may be toxic to ethanol-induced liver disease patients [ 16 ].…”
Section: Vitamin Amentioning
confidence: 99%