Troponina T positiva em paciente chagásico com taquicardia ventricular sustentada e cinecoronariografia sem lesões obstrutivas

Machado, Maurício de Nassau; Suzuki, F; Mouco, Osana Costa C.; Hernandes, Mauro Esteves; Lemos, Maria Angélica; Maia, Lília Nigro

doi:10.1590/s0066-782x2005000200019

Cited by 7 publications

(4 citation statements)

References 10 publications

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“…In spite of the optimal accuracy to detect myocardial injury in ACS, the troponins can rise in a series of nonatherosclerotic heart diseases [4, 5]. In situations in which the troponin levels are elevated and the clinical state is not consistent with MI, other causes of myocardial injury must be considered with, as a last resort, the possibility of a false-positive result [5, 11].…”

Section: Discussionmentioning

confidence: 99%

“…In situations in which the troponin levels are elevated and the clinical state is not consistent with MI, other causes of myocardial injury must be considered with, as a last resort, the possibility of a false-positive result [5, 11]. …”

Section: Discussionmentioning

confidence: 99%

“…Numerous conditions have been described as causes of myocardial injury not related to atherosclerotic disease including congestive heart disease, sepsis, pulmonary emboli, thoracic contusion, coronary emboli caused by endocarditis, neoplasms, prosthetic valves, inflammatory processes including viral infection by Coxsackie B, coronary stenosis induced by radiation, cocaine use and strenuous physical exercise [1, 4, 5,12,13,14,15]. …”

Section: Discussionmentioning

confidence: 99%

“…On the other hand, there are few published reports on patients with high troponin levels secondary to supraventricular arrhythmias [1]. Such conditions reflect the importance of the diagnostic differentiation when attending patients suffering from chest pain in the emergency room [5]. We describe the case of a 49-year-old previously healthy woman who was admitted with chest discomfort and supraventricular tachycardia and with high cardiac troponin T (TnT) levels but with a normal coronary angiography.…”

Section: Introductionmentioning

confidence: 99%

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Elevated Troponin Levels after Prolonged Supraventricular Tachycardia in Patient with Normal Coronary Angiography

et al. 2006

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The European Society of Cardiology and the American College of Cardiology redefined the concept of myocardial infarction in the presence of highly positive markers of myocardial injury associated with at least one of the following: ischemic symptoms; development of pathologic Q waves on the ECG or ECG changes indicative of ischemia (positive or negative deviation of the ST segment), making troponins one of the most important aspects in the evaluation and stratification of patients with chest pain in the emergency room. However, although troponin gives excellent accuracy in the identification of myocardial necrosis, it is known that it can also be elevated in a series of nonatherosclerotic heart diseases. We present the case of a 49-year-old female patient admitted to the Chest Pain Unit with a history of supraventricular tachycardia associated with chest discomfort, nausea and diaphoresis. During risk stratification, the patient presented with a high serum troponin T level (0.143 ng/ml) but with a normal coronary angiography.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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Elevated Troponin Levels after Prolonged Supraventricular Tachycardia in Patient with Normal Coronary Angiography

et al. 2006

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Chronic Chagas’ heart disease: a disease on its way to becoming a worldwide health problem: epidemiology, etiopathology, treatment, pathogenesis and laboratory medicine

et al. 2010

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Chagas' disease, caused by Trypanosoma cruzi infection, is ranked as the most serious parasitic disease in Latin America. Nearly 30% of infected patients develop life-threatening complications, and with a latency of 10-30 years, mostly Chagas' heart disease which is currently the major cause of morbidity and mortality in Latin America, enormously burdening economic resources and dramatically affecting patients' social and labor situations. Because of increasing migration, international tourism and parasite transfer by blood contact, intrauterine transfer and organ transplantation, Chagas' heart disease could potentially become a worldwide problem. To raise awareness of this problem, we reflect on the epidemiology and etiopathology of Chagas' disease, particularly Chagas' heart disease. To counteract Chagas' heart disease, in addition to the general interruption of the infection cycle and chemotherapeutic elimination of the infection agent, early and effective causal or symptomatic therapies would be indispensable. Prerequisites for this are improved knowledge of the pathogenesis and optimized patient management. From economic and logistics viewpoints, this last prerequisite should be performed using laboratory medicine tools. Consequently, we first summarize the mechanisms that have been suggested as driving Chagas' heart disease, mainly those associated with the presence of autoantibodies against G-protein-coupled receptors; secondly, we indicate new treatment strategies involving autoantibody apheresis and in vivo autoantibody neutralization; thirdly, we present laboratory medicine tools such as autoantibody estimation and heart marker measurement, proposed for diagnosis, risk assessment and patient guidance and lastly, we critically reflect upon the increase in inflammation and oxidative stress markers in Chagas' heart disease.

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Troponin-I elevation in a young man with arrhythmogenic right ventricular dysplasia/cardiomyopathy

Kostis

Tedford

Miller

et al. 2008

J Interv Card Electrophysiol

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Ventricular arrhythmias occur frequently in patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy (ARVD/C) as well as those with ischemic heart disease. We present the case of a 29-year-old man with ARVD/C and multiple episodes of symptomatic ventricular tachycardia terminated by implantable cardioverter defibrillator (ICD) discharges. Phasic elevation of troponin-I prompted repeated coronary angiograms, all of which were normal. The patient was successfully treated with radiofrequency ablation. This case illustrates that ARVD/C may result in elevated cardiac enzymes in the absence of coronary artery disease.

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Troponina T positiva em paciente chagásico com taquicardia ventricular sustentada e cinecoronariografia sem lesões obstrutivas

Cited by 7 publications

References 10 publications

Elevated Troponin Levels after Prolonged Supraventricular Tachycardia in Patient with Normal Coronary Angiography

Elevated Troponin Levels after Prolonged Supraventricular Tachycardia in Patient with Normal Coronary Angiography

Chronic Chagas’ heart disease: a disease on its way to becoming a worldwide health problem: epidemiology, etiopathology, treatment, pathogenesis and laboratory medicine

Troponin-I elevation in a young man with arrhythmogenic right ventricular dysplasia/cardiomyopathy

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