2020
DOI: 10.1111/bjd.19115
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Abstract: Background Human hair is highly responsive to stress, and human scalp hair follicles (HFs) contain a peripheral neuroendocrine equivalent of the systemic hypothalamic-pituitary-adrenal (HPA) stress axis. Androgenetic alopecia (AGA) is supposed to be aggravated by stress. We used corticotropin-releasing hormone (CRH), which triggers the HPA axis, to induce a stress response in human ex vivo male AGA HFs. Caffeine is known to reverse testosterone-mediated hair growth inhibition in the same hair organ culture mod… Show more

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Cited by 17 publications
(19 citation statements)
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“…This time, Fischer et al . have demonstrated the protective role of caffeine against all the aforementioned CRH‐induced events in AGA HFs, further supporting a potential benefit of caffeine in the management of AGA 6 …”
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confidence: 68%
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“…This time, Fischer et al . have demonstrated the protective role of caffeine against all the aforementioned CRH‐induced events in AGA HFs, further supporting a potential benefit of caffeine in the management of AGA 6 …”
mentioning
confidence: 68%
“…In addition, CRH increased non‐HPA axis‐related stress mediators, including substance P and p75 neurotrophin receptor. These findings are in favour of an aggravative role of stress in AGA 6 …”
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confidence: 84%
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