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Cited by 2,402 publications
(1,052 citation statements)
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References 14 publications
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“…As previously reported, [4] the addition of norepinephrine (NE) results in a large and time-dependent Mg 2+ efflux. A qualitatively similar efflux was observed after stimulation with staurosporin, an inhibitor of" PKC [26]. By contrast, the addition of carbachol stimulates Mg 2+ uptake by the myocytes.…”
Section: Figuresupporting
confidence: 67%
See 1 more Smart Citation
“…As previously reported, [4] the addition of norepinephrine (NE) results in a large and time-dependent Mg 2+ efflux. A qualitatively similar efflux was observed after stimulation with staurosporin, an inhibitor of" PKC [26]. By contrast, the addition of carbachol stimulates Mg 2+ uptake by the myocytes.…”
Section: Figuresupporting
confidence: 67%
“…Table I confirms the involvement of PKC in the modulation of Mg 2+ uptake. Myocytes incubated in the presence of the diacylglycerol analogs OAG and SAG or in the presence of the phorbol esters TPA and PDBU, agents directly stimulating the activity of PKC [26][27][28][29][30], accumulate Mg-'+ from the medium similar to that observed in the presence of carbachol. By contrast, the addition of the PKC inhibitor H7 [3 l] stimulates a Mg -~* efflux from myocytes quantitatively similar to that observed with norepinephrine (Fig.…”
Section: Figurementioning
confidence: 97%
“…3 A and B) and no greater inhibition was observed at a higher dose of 25 nM in either tissue (data not shown). As PKI-STSP selectively inhibits PKC at the lower concentration of 5 nM and various protein kineses, including PKA, PKG, and CaMKII, at the higher concentrations of 25 nM (Tamaoki et al, 1986), our results indicate that PKC is likely involved in sustained activation of ADP-ribosyl cyclase. This was confirmed by using a more specific PKC inhibitor, calphostin C (100 nM), in both tissues challenged by these two concentrations (Fig.…”
Section: Inhibition Of Ot-induced Activation Of the Adp-ribosyl Cyclasementioning
confidence: 58%
“…31 To test whether c-Myc-deficient B lymphocytes were also resistant to a different apoptotic stimuli, we treated them with staurosporine. Sorted B lymphocytes were activated with anti-CD40 antibody or anti-CD40 plus interleukin 4 and culture for 48 h before adding staurosporine.…”
Section: Resultsmentioning
confidence: 99%