The challenges facing neurosurgical healthcare in India include accessibility, affordability, infrastructure, medical malpractice, and training and education. The lack of infrastructure and shortage of trained professionals are significant issues impacting the quality of care provided to patients. To address these challenges, there is a need for increased investment in facilities, expanding access to specialized equipment, increasing the number of trained staff, and improving the overall quality of healthcare facilities. Collaboration between government, private sector, and non-profit organizations is also necessary to ensure that patients receive comprehensive, high-quality care, regardless of their location or ability to pay. Additionally, addressing the shortage of trained neurosurgeons, neurologists, and neuroanesthesiologist in India is crucial to meet the growing demand for their services.
Introduction Decompressive craniectomies have been performed in settings with raised intracranial pressure (ICP) after severe traumatic brain injury (TBI). A decompressive craniectomy (DC) is an important salvage procedure for intracranial hypertension. The changes in the intracranial microenvironment after a primary DC are significant in terms of the neurological outcome in the postoperative period. Materials and methods The study comprised 68 patients with severe TBIs who were undergoing primary DC; of these, 59% were male. Recorded data include demographic profiles, clinical features, and cranial computed tomography (CT) scans. All patients underwent a primary unilateral DC with augmentation duraplasty. Intracranial pressure was recorded in the first 24 hours at regular intervals, and the outcome was recorded using the Extended Glasgow Outcome Scale (GOS-E) at two-week and two-month intervals. Results Road traffic accidents (RTAs) are the most common cause of severe TBIs. Imaging studies and intraoperative findings suggest that acute subdural hematomas (SDHs) are the most common pathology leading to high ICP in the postoperative period. Mortality was strongly statistically associated with high ICP values postoperatively at all intervals. The average ICP for the patients who died was 11.871 mmHg higher than the patients who survived (p=0.0009). The Glasgow Coma Scale (GCS) at the time of admission is positively correlated with the neurological outcome at two weeks and two months, with a Pearson correlation coefficient of 0.4190 and 0.4235, respectively. There is a strong negative correlation between ICP in the postoperative period and the neurological outcome at two weeks and two months (Pearson correlation coefficients are −0.828 and −0.841, respectively). Conclusion The results indicate that RTAs are the most common cause of severe TBIs, and acute SDHs are the most common pathology leading to high ICP after the surgery. ICP values in the postoperative period have a strong negative correlation with survival and neurological outcome. Preoperative GCS and postoperative ICP monitoring are important methods of prognostication and planning further management.
This case report describes a 30-year-old woman who developed an acute spontaneous subdural hematoma (SDH) after receiving intraspinal anesthesia for a cesarean section, presenting with only headache as an initial symptom. The purpose of the report is to emphasize the importance of considering acute spontaneous SDH as a potential complication of intraspinal anesthesia in patients presenting with headache, even in the absence of other neurological deficits, and the need for prompt recognition and management of this condition, as early intervention can significantly improve outcomes. The report also highlights the importance of informed consent and patient education about the potential risks and benefits of different types of anesthesia during cesarean section. The discussion includes the pathophysiology of subdural hematoma after spinal anesthesia, potential causes of severe headache, and the importance of distinguishing between neurological symptoms of intracranial hypotension, post-dural puncture headache (PDPH), and subdural hematoma. The patient underwent burr hole evacuation after the subdural hematoma converted completely to chronic, with no neurological abnormality or recurrence till now.
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