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This prospective longitudinal investigation examined early mother-child interaction as a predictor of childrenThe emergence of self-regulation is one of the most significant developmental landmarks of the early childhood period

Assessed the antecedents of individual differences in children's cognitive/language competence at age 24 mo using multivariate methods at ages 6, 13, and 24 mo in 121 Ss. Assessments included detailed observations of mother–children interaction, standardized tests of child cognitive development (including the Home Observation for Measurement of the Environment and the Bayley Mental Scale of Infant Development), and examiner and maternal ratings of child sociability. There were meaningful patterns of continuity in the child and especially mother behavior factors across ages, and mother–child warm, verbal interactions at each age were associated with a composite index of child competence at 24 mo. A path analysis showed that altogether, 40% of the variance in competence outcome was explained by the 4 variables in the model (interaction at 3 ages and SES). The analysis also showed that the original bivariate correlation between interaction at 6 mo and later competence could be explained by continuity in interaction qualities at 6 and 24 mo rather than as a direct effect. However, the 13-mo interaction showed both a direct path to competence and an indirect one via age 24-mo interaction. SES had a modest significant correlation with competence, but in the path analysis this was dissipated in nonsignificant paths to the interaction variables. Partial correlations suggested that child developmental competence and sociability at earlier ages did not mediate the relationships between mother–infant interaction and later child competence. (34 ref)

This study tested (a) the hypothesis that Parkinson's disease (PD), thought to result in disturbed neuronal outflow from the striatum, leads to circumscribed deficits in cognitive functions presumed to be dependent on the functional integrity of the frontal lobes and (b) whether such deficits could account for previously reported memory and visuoperceptual difficulties in PD. Nondemented PD patients (n = 19) were demographically matched to 19 normal elderly control subjects. Three categories of tests were given: (a) tests sensitive to frontal system dysfunction, (b) tests of learning and memory, and (c) tests of visuoperceptual and visuoconstructive skills. Nondemented PD patients demonstrated selective deficits on frontal system tasks: tests of learning and memory and of visuoperceptual and visuoconstructive skill were not significantly impaired once performance on the frontal related tasks was statistically covaried. Results are consistent with the striatofrontal outflow model of neuropsychological impairment in nondemented PD patients.

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