The main objective of this research was to evaluate the efficacy of intermittent hypoxic training (IHT) on anaerobic and aerobic capacity and swimming performance in well-trained swimmers. Sixteen male swimmers were randomly divided into a hypoxia (H) group (n = 8), which trained in a normobaric hypoxia environment, and a control (C) group (n = 8), which exercised under normoxic conditions. However, one participant left the study without explanation. During the experiment group H trained on land twice per week in simulated hypoxia (FiO2 = 15.5%, corresponding to 2,500 m a.s.l); however, they conducted swim training in normoxic conditions. Group C performed the same training program under normoxic conditions. The training program included four weekly microcyles, followed by three days of recovery. During practice sessions on land, the swimmers performed 30 second sprints on an arm-ergometer, alternating with two minute high intensity intervals on a lower limb cycle ergometer. The results showed that the training on land caused a significant (p<0.05) increase in absolute maximal workload (WRmax) by 7.4% in group H and by 3.2% in group C and relative values of VO2max by 6.9% in group H and 3.7% in group C. However, absolute values of VO2max were not significantly changed. Additionally, a significant (p<0.05) increase in mean power (Pmean) during the first (11.7%) and second (11.9%) Wingate tests was only observed in group H. The delta values of lactate concentration (ΔLA) after both Wingate tests were significantly (p<0.05) higher in comparison to baseline levels by 28.8% in group H. Opposite changes were observed in delta values of blood pH (ΔpH) after both Wingate tests in group H, with a significant decrease in values of ΔpH by 33.3%. The IHT caused a significant (p<0.05) improvement in 100m and 200m swimming performance, by 2.1% and 1.8%, respectively in group H. Training in normoxia (group C), resulted in a significant (p<0.05) improvement of swimming performance at 100m and 200m, by 1.1% and 0.8%, respectively. In conclusion, the most important finding of this study includes a significant improvement in anaerobic capacity and swimming performance after high-intensity IHT. However, this training protocol had no effect on absolute values of VO2max and hematological variables.
The aim of this study was to assess the impact of a 6-week specialized training program aimed at strengthening core muscles to improve the effectiveness of selected elements of a swimming race on a group of Polish swimmers. Sixteen male national level swimmers (21.6 ± 2.2 years) participated in the research. The competitors were randomly assigned to 1 of 2 groups before the data collection process: an experimental (EG, n = 8) and a control (CG, n = 8) group. Both groups of swimmers underwent the same training program in the water environment (volume and intensity), while swimmers from the EG additionally performed specific core muscle training. The task of the swimmers was an individual front crawl swim of 50 m, during which the kinematic parameters of the start jump, turn and swimming techniques were recorded using a video camera system. In both groups, a minor increase in the flight phase was observed at the start (EG = 0.06 m, 1.8%; p = 0 . 088 ; CG = 0.08 m, 2.7%; p = 0 . 013 ). The time to cover a distance of 5 m after the turn and the recorded average speed in swimming this distance for the EG statistically significantly improved by 0.1 s (-28.6%; p < 0 . 001 ) and 3.56 m∙s -1 (23.2%; p = 0 . 001 ), respectively. In the EG, a statistically significant improvement in 50 m front crawl swimming performance of 0.3 s (-1.2%, p = 0 . 001 ) was observed. The results of the research show that the implementation of isolated strengthening of the stabilizing muscles seems to be a valuable addition to the standard training of swimmers.
The main objective of this study was to examine the chronic effect of simulated intermittent normobaric hypoxia on blood antioxidant defence capacity in swimmers. The study included 14 male and 14 female competitive swimmers performing part of land training under simulated intermittent normobaric hypoxia (O2 = 15.5%) or in normoxia. Land interval training took place twice per week, with a total of 8 training units during the study, performed with individualized intensity. The activities of blood antioxidant enzymes did not change significantly during the first and last training unit in the hypoxic and normoxic group. However, when comparing individual variables a significant effect of exercise was observed on GPx an CAT activities, whereas training units significantly differentiated GPx and GR activities. The oxygen conditions and gender had a significant influence on CAT activity. The total antioxidant capacity was not significantly affected. Only in male swimmers from the hypoxic group did the training significantly increase resting levels of MDA. In conclusion, training in normobaric hypoxia was not an adequate stimulus for the excessive response of the antioxidant defence system, despite increased oxidative stress in these conditions.
This study aims to evaluate the effects of a prolonged transition period (detraining) on the physical capacity, power, and speed parameters of elite swimmers. Fourteen swimmers (seven females and seven males) aged 20.4 ± 1.7 years participated in the study. The athletes were subjected to two rounds of identical tests at 12-week intervals during the detraining period (DP), which consisted of an evaluation of the athletes’ body weight and composition, a measurement of the power of their lower limbs (Keiser squat, countermovement jump (CMJ), akimbo countermovement jump (ACMJ)) and upper limbs (Keiser arms) on land, and 20-m swimming using the legs only (Crawl Legs test), arms only (Crawl Arms test), and full stroke (Front Crawl test). An analysis of variance revealed a significant effect of the main factor, Gender, on all the measured parameters, while for the factor Detraining, except for Front Crawl (W) (F = 4.27, p = 0.061), no significant interaction effect (Gender × Detraining) was revealed. Among both the male and the female participants, a reduction in lactate-threshold swimming speed (LT Dmax) and a reduction in swimming speed and power on the Crawl Arms, Crawl Legs, and Front Crawl tests was observed after 12 weeks. There were also statistically significant reductions in ACMJ and CMJ jump height and upper-limb power (Keiser squat) among the female and male swimmers. There were no significant changes in body weight or body composition. The study showed a clear deterioration in results for most of the parameters, both for those measured on land and for those measured in water.
Objectives: Swimming is one of the most popular forms of physical activity. Pool water is cleaned with chlorine, which -in combination with compounds contained in water -could form chloramines and trichloromethane in the swimmer's lungs. The aim of the present study was to examine the effect of swimming training in an indoor pool on the composition of swimmers' respiratory phase metabolomics, and develop a system to provide basic information about its impact on the swimmer's airway mucosa metabolism, which could help to assess the risk of secondary respiratory tract diseases i.e. sport results, condition, and health including lung acute and chronic diseases). Design: A group of competitive swimmers participated in the study and samples of their respiratory phase before training, immediately after training, and 2 h after training were assessed. Methods: Sixteen male national and international-level competitive swimmers participated in this study. Respiratory phase analysis of the indoor swimming pool swimmers was performed. Gas chromatography combined with mass spectrometry (GCMS) was used in the measurements. All collected data were transferred to numerical analysis for trends of tracking and mapping. The breathing phase was collected on special porous material and analyzed using GCMS headspace. Results: The obtained samples of exhaled air were composed of significantly different metabolomics when compared before, during and after exercise training. This suggests that exposition to indoor chlorine causes changes in the airway mucosa.(Continued on next page)
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