Organics-capped ZnSe quantum dots were synthesized by a colloidal chemical approach using ZnO and Se powder as precursors. The photoluminescence of the specimens showed strong white emission ͑ϳ200 nm full width at half maximum͒ in the visible range under ambient conditions. The white emission was attributed to the mixing of blue emission of ZnSe nanocrystals exhibiting quantum confinement effect with green-red emission of radiative deep levels from ZnSe surface strained lattice. Based on organic-capped ZnSe quantum dots, the white-light-emitting diodes were fabricated using a near-UV InGaN chip as the excitation source. The diodes emitted white light with CIE chromaticity coordinates of ͑0.38 and 0.41͒ and show great potential for use in lighting applications.

Colloidal ZnSe quantum dots were successfully synthesized from ZnO in a lauric acid/hexadecylamine mixture. X-ray diffraction patterns indicated that the ZnSe quantum dots possess a wurtzite structure. Transmission electron microscope images of the quantum dots showed that the average diameter are in the range of 25∼60 Å. The size-dependent photoluminescence was controlled from 400 to 440 nm with the quantum yields of 6∼10% at room temperature. After passivation with 1.8 monolayer of ZnS overcoat by a traditional two-step method, the quantum yields of ZnSe/ZnS QDs are increased ∼4.5-fold (quantum yield ∼32%). An in-situ method of overcoating was done by directly injecting TOPS into the flask containing ZnSe, ZnO/lauric acid/ hexadecylamine, and TOPSe. The photoluminescence quantum yields were improved ∼3.8-fold after introduction of 1.6 monolayer ZnSeS overcoat to the ZnSe QDs.

Halide-driven partial and complete exchange of Pb2+ and Sn2+ occurred deliberately. It is found that more Pb ions originated from the Pb–Br species to form Pb–OA complexes. Such halide exchange reaction opened the octahedral structure of CsPbBr3 NCs.

A promising method has been demonstrated to fabricate quantum dot (QD)-converted full-color micro-light emitting diodes (LEDs) by inkjet printing (IJP) instead of the mass transfer of three red-green-blue (RGB) color chips. By introducing an additional medium, that is, NaCl into a formulated ink, QD deposition is manipulated by the NaCl-QD adhesive force and the capillary flow inside the liquid drop via varying the substrate hydrophobicity, which enabled spontaneous self-encapsulation of QDs in a single NaCl crystal. An RGB QD@NaCl array with a small pixel size and uniform size distribution (diameter = 3.74 ± 0.5 μm) is obtained in the IJP process, which demonstrated a full-color micro-LED display with a color gamut of approximately 110% of the National Television System Committee (NTSC) standard.

Particles present in diesel exhaust have been proposed as a significant contributor to the development of acute and chronic lung diseases, including respiratory infection and allergic asthma. Nanoceria (CeO2 nanoparticles) are used to increase fuel efficiency in internal combustion engines, are present in exhaust fumes, and could affect cells of the airway. Components from the environment such as biologically derived proteins, carbohydrates, and lipids can form a dynamic layer, commonly referred to as the "protein corona" which alters cellular nanoparticle interactions and internalization. Using confocal reflectance microscopy, we quantified nanoceria uptake by lung-derived cells in the presence and absence of a serum-derived protein corona. Employing mass spectrometry, we identified components of the protein corona, and demonstrated that the interaction between transferrin in the protein corona and the transferrin receptor is involved in mediating the cellular entry of nanoceria via clathrin-mediated endocytosis. Furthermore, under these conditions nanoceria does not affect cell growth, viability, or metabolism, even at high concentration. Alternatively, despite the antioxidant capacity of nanoceria, in serum-free conditions these nanoparticles induce plasma membrane disruption and cause changes in cellular metabolism. Thus, our results identify a specific receptor-mediated mechanism for nanoceria entry, and provide significant insight into the potential for nanoparticle-dependent toxicity.