BackgroundThe treatment of coronary artery disease (CAD) seeks to reduce or prevent its complications and decrease morbidity and mortality. For certain subgroups of patients, coronary artery bypass graft surgery (CABG) may accomplish these goals. The objective of this study was to assess the pulmonary function in the CABG postoperative period of patients treated with a physiotherapy protocol.MethodsForty-two volunteers with an average age of 63 ± 2 years were included and separated into three groups: healthy volunteers (n = 09), patients with CAD (n = 9) and patients who underwent CABG (n = 20). Patients from the CABG group received preoperative and postoperative evaluations on days 3, 6, 15 and 30. Patients from the CAD group had evaluations on days 1 and 30 of the study, and the healthy volunteers were evaluated on day 1. Pulmonary function was evaluated by measuring forced vital capacity (FVC), maximum expiratory pressure (MEP) and Maximum inspiratory pressure (MIP).ResultsAfter CABG, there was a significant decrease in pulmonary function (p < 0.05), which was the worst on postoperative day 3 and returned to the preoperative baseline on postoperative day 30.ConclusionPulmonary function decreased after CABG. Pulmonary function was the worst on postoperative day 3 and began to improve on postoperative day 15. Pulmonary function returned to the preoperative baseline on postoperative day 30.
Introduction Imbalance in autonomic cardiovascular function increases the risk for sudden death in patients with coronary artery disease (CAD), but the time course of the impact of coronary artery bypass grafting (CABG) on autonomic function has been little studied. Thus, the purpose of the present study was to determine the effects of the CABG on the cardiovascular autonomic function. Methods Patients undergoing CABG (n = 13) and two matched control groups (patients with CAD who refused surgical treatment [n = 9], and healthy volunteers [n = 9]) underwent a prospective longitudinal study consisting of autonomic evaluation before and after (3,6,15, 30, 60, and 90 days) surgery, including measurement of heart rate variability (HRV), respiratory sinus arrhythmia (RSA), and Valsalva maneuver. Results After CABG there was a decrease in, and a later recovery of, (1) the HRV in the time domain and in the frequency domain, (2) RSA, and (3) Valsalva maneuver. Conclusions CABG caused an impairment, reversible after 60 days, of cardiovascular autonomic function, with a maximal decrease on about the sixth day after surgery.
The purpose of this study was to determine the effect of respiratory muscle fatigue
on intercostal and forearm muscle perfusion and oxygenation in patients with heart
failure. Five clinically stable heart failure patients with respiratory muscle
weakness (age, 66±12 years; left ventricle ejection fraction, 34±3%) and nine matched
healthy controls underwent a respiratory muscle fatigue protocol, breathing against a
fixed resistance at 60% of their maximal inspiratory pressure for as long as they
could sustain the predetermined inspiratory pressure. Intercostal and forearm muscle
blood volume and oxygenation were continuously monitored by near-infrared
spectroscopy with transducers placed on the seventh left intercostal space and the
left forearm. Data were compared by two-way ANOVA and Bonferroni correction.
Respiratory fatigue occurred at 5.1±1.3 min in heart failure patients and at 9.3±1.4
min in controls (P<0.05), but perceived effort, changes in heart rate, and in
systolic blood pressure were similar between groups (P>0.05). Respiratory fatigue
in heart failure reduced intercostal and forearm muscle blood volume (P<0.05)
along with decreased tissue oxygenation both in intercostal (heart failure,
-2.6±1.6%; controls, +1.6±0.5%; P<0.05) and in forearm muscles (heart failure,
-4.5±0.5%; controls, +0.5±0.8%; P<0.05). These results suggest that respiratory
fatigue in patients with heart failure causes an oxygen demand/delivery mismatch in
respiratory muscles, probably leading to a reflex reduction in peripheral limb muscle
perfusion, featuring a respiratory metaboreflex.
Objetivo: Correlacionar poluição atmosférica com hospitalizações de idosos em Nova Iguaçu por doenças pulmonares entre 2007 a 2016. Metodologia: Estudo observacional, retrospectivo. O poluente analisado foi o Material Particulado (PM10). Os dados de qualidade do ar foram fornecidos pelo (INEA). Dados de hospitalização pelo (AIH). Foram selecionadas doenças do aparelho respiratório pelo CID X e subcapítulos J. A análise estatística foi realizada pelo SPSS IBM25 com dados expressos em média, desvio padrão, mediana e intervalo de confiança de 95%. Utilizou-se o teste t para amostras independentes, a diferença estatística foi dada pelo p<0,05. A relação entre poluição do ar e hospitalizações utilizou o coeficiente de correlação de Pearson. Resultados: As médias mensais de PM10 e hospitalizações foram, respectivamente, 74,7 mg/m3 (±16,2), (IC=63,8-87,1); 40,6 (±13,5) (IC=37,5 – 43,5). Os homens representaram (52,4%), que totalizaram 2.239 internações em 10 anos, em que 2015 foi o ano com maior índice de hospitalizações (615). A doença mais incidente foi o J18 com média anual de 102,7. A pneumonia viral acometeu três vezes mais mulheres (p<0,042). A correlação de Pearson entre PM10 e hospitalizações (R=-0,230). Conclusão: O sexo mais acometido entre os idosos foi o masculino. Os agravos pulmonares de maior hospitalização foram as pneumonias, insuficiência respiratória e DPOC. Apesar da não conformidade dos níveis de PM10 em 10 anos houve redução de 60% das concentrações. Os meses entre maio e agosto demonstraram as maiores elevações de PM10 e hospitalizações. A correlação mensal entre hospitalizações de idosos e PM10 foi negativa e fraca.
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.