Augmented contraction at maximal dobutamine depends not only on increased myocardial blood flow but also on tethering, metabolic, and beta-blocker status. Furthermore, impaired flow reserve does not preclude a normal response to maximal dobutamine, since blood flow need not increase greatly to meet demand.
Este texto está dedicado a Cecilia Méndez Gastelumendi, a quien agradezco por su orientación y atinados comentarios. Una versión preliminar de este texto fue leída en julio del 2006, en el 52º Congreso Internacional de Americanistas en Sevilla (España).
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